长时程增强：急切寻求解决方案。 - Suppr

Suppr

超能文献

LTP: desperately seeking resolution.

作者信息

Malinow R

机构信息

Cold Spring Harbor Laboratory, NY 11724.

出版信息

Science. 1994 Nov 18;266(5188):1195-6. doi: 10.1126/science.7973700.

DOI:10.1126/science.7973700

PMID:7973700

Abstract

摘要

相似文献

LTP: desperately seeking resolution.长时程增强：急切寻求解决方案。

Science. 1994 Nov 18;266(5188):1195-6. doi: 10.1126/science.7973700.

Long-term potentiation involves increases in the probability of neurotransmitter release.长时程增强作用涉及神经递质释放概率的增加。

Proc Natl Acad Sci U S A. 1997 May 27;94(11):5888-93. doi: 10.1073/pnas.94.11.5888.

Long-term potentiation--a decade of progress?长期增强作用——十年进展？

Science. 1999 Sep 17;285(5435):1870-4. doi: 10.1126/science.285.5435.1870.

[Advancement in mechanisms of long-term potentiation].[长期增强作用机制的进展]

Sheng Li Ke Xue Jin Zhan. 2001 Oct;32(4):298-301.

Activation of postsynaptically silent synapses during pairing-induced LTP in CA1 region of hippocampal slice.海马切片CA1区配对诱导的长时程增强过程中突触后沉默突触的激活。

Nature. 1995 Jun 1;375(6530):400-4. doi: 10.1038/375400a0.

Long-term potentiation: evidence against an increase in transmitter release probability in the CA1 region of the hippocampus.长期增强效应：反对海马体CA1区神经递质释放概率增加的证据。

Science. 1994 Sep 23;265(5180):1888-92. doi: 10.1126/science.7916483.

[Long-term potentiation of AMPA- and NMDA-components of minimal postsynaptic currents in CA1 area of the rat hippocampus].[大鼠海马体CA1区最小突触后电流中AMPA和NMDA成分的长期增强]

Ross Fiziol Zh Im I M Sechenova. 2001 Apr;87(4):448-58.

Non-Hebbian properties of long-term potentiation enable high-capacity encoding of temporal sequences.长时程增强的非赫布特性可实现时间序列的高容量编码。

Proc Natl Acad Sci U S A. 1994 Oct 11;91(21):10104-8. doi: 10.1073/pnas.91.21.10104.

Presynaptic changes during mossy fibre LTP revealed by NMDA receptor-mediated synaptic responses.NMDA受体介导的突触反应揭示的苔藓纤维长时程增强期间的突触前变化。

Nature. 1995 Jul 20;376(6537):256-9. doi: 10.1038/376256a0.

Leitmotifs in the biochemistry of LTP induction: amplification, integration and coordination.长时程增强（LTP）诱导的生物化学中的主导主题：放大、整合与协调。

J Neurochem. 2001 May;77(4):961-71. doi: 10.1046/j.1471-4159.2001.00321.x.

引用本文的文献

Mechanistic Understanding of the Phosphorylation-Induced Conformational Rigidity at the AMPA Receptor C-terminal Domain.对AMPA受体C端结构域磷酸化诱导的构象刚性的机制理解

ACS Omega. 2019 Aug 20;4(10):14211-14218. doi: 10.1021/acsomega.9b01384. eCollection 2019 Sep 3.

Phosphorylation Induces Conformational Rigidity at the C-Terminal Domain of AMPA Receptors.磷酸化诱导 AMPA 受体 C 端结构域构象刚性。

J Phys Chem B. 2019 Jan 10;123(1):130-137. doi: 10.1021/acs.jpcb.8b10749. Epub 2018 Dec 27.

Regulation of GluA1 α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor function by protein kinase C at serine-818 and threonine-840.蛋白激酶 C 在丝氨酸 818 和苏氨酸 840 位点对 GluA1 α-氨基-3-羟基-5-甲基-4-异恶唑丙酸受体功能的调节。

Mol Pharmacol. 2014 Apr;85(4):618-29. doi: 10.1124/mol.113.091488. Epub 2014 Jan 22.

Presynaptic long-term plasticity.突触前长期可塑性

Front Synaptic Neurosci. 2013 Oct 17;5:8. doi: 10.3389/fnsyn.2013.00008.

Dendritic D-type potassium currents inhibit the spike afterdepolarization in rat hippocampal CA1 pyramidal neurons.树突状D型钾电流抑制大鼠海马CA1锥体神经元的锋电位后去极化。

J Physiol. 2007 May 15;581(Pt 1):175-87. doi: 10.1113/jphysiol.2006.127068. Epub 2007 Feb 22.

Multiple, developmentally regulated expression mechanisms of long-term potentiation at CA1 synapses.CA1突触处长期增强的多种、受发育调控的表达机制。

J Neurosci. 2004 May 26;24(21):4903-11. doi: 10.1523/JNEUROSCI.0170-04.2004.

Perceptual correlates of nociceptive long-term potentiation and long-term depression in humans.人类伤害性长时程增强和长时程抑制的知觉相关因素。

J Neurosci. 2004 Jan 28;24(4):964-71. doi: 10.1523/JNEUROSCI.1222-03.2004.

NMDA antagonists in the superior colliculus prevent developmental plasticity but not visual transmission or map compression.上丘中的NMDA拮抗剂可阻止发育可塑性，但不影响视觉传导或图谱压缩。

J Neurophysiol. 2001 Sep;86(3):1179-94. doi: 10.1152/jn.2001.86.3.1179.

Silent synapses in the developing rat visual cortex: evidence for postsynaptic expression of synaptic plasticity.发育中大鼠视觉皮层的沉默突触：突触可塑性的突触后表达证据

J Neurosci. 1998 Nov 1;18(21):8863-74. doi: 10.1523/JNEUROSCI.18-21-08863.1998.

Ca2+/calmodulin-dependent protein kinase II phosphorylation of the presynaptic protein synapsin I is persistently increased during long-term potentiation.在长时程增强过程中，突触前蛋白突触素I的钙/钙调蛋白依赖性蛋白激酶II磷酸化持续增加。

Proc Natl Acad Sci U S A. 1996 Dec 24;93(26):15451-6. doi: 10.1073/pnas.93.26.15451.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验