Clinical improvement in psoriasis during dithranol/UVB therapy does not correspond with a decrease in elevated serum soluble ICAM-1 levels.

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作者信息

Kowalzick L, Bildau H, Neuber K, Köhler I, Ring J

机构信息

Universitäts-Hautklinik Hamburg-Eppendorf, Hamburg, Germany.

出版信息

Arch Dermatol Res. 1993;285(4):233-5. doi: 10.1007/BF00372016.

DOI:10.1007/BF00372016

PMID:8102047

Abstract

摘要

相似文献

Clinical improvement in psoriasis during dithranol/UVB therapy does not correspond with a decrease in elevated serum soluble ICAM-1 levels.

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Elevated serum-soluble ELAM-1 levels in patients with severe plaque-type psoriasis.重度斑块型银屑病患者血清可溶性ELAM-1水平升高。

Arch Dermatol Res. 1994;286(7):414-6. doi: 10.1007/BF00371802.

Psoriasis treatment: faster clearance when UVB-dithranol is combined with topical clobetasol propionate.银屑病治疗：当紫外线B-地蒽酚与外用丙酸氯倍他索联合使用时，清除速度更快。

Dermatologica. 1986;172(3):164-8. doi: 10.1159/000249323.

Calcitriol vs. dithranol in combination with narrow-band ultraviolet B (311 nm) in psoriasis.骨化三醇与地蒽酚联合窄谱紫外线B（311nm）治疗银屑病的对比研究

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Targeted 307 nm UVB-excimer light vs. topical dithranol in psoriasis.靶向307纳米准分子紫外线与局部用蒽林治疗银屑病的对比

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Clinical studies with a novel dithranol formulation (Micanol) in combination with UVB at day-care centres.在日间护理中心使用新型地蒽酚制剂（米卡诺）联合紫外线B进行的临床研究。

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[Study on the efficacy of a combined treatment of psoriasis with dithranol and UV-B (selective ultraviolet-phototherapy)].[地蒽酚与UV-B联合治疗银屑病（选择性紫外线光疗法）的疗效研究]

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引用本文的文献

sICAM-1, sIL-2R and beta 2-microglobulin serum levels in patients affected with psoriasis: relationship with disease severity.银屑病患者血清中可溶性细胞间黏附分子-1、可溶性白细胞介素-2受体和β2-微球蛋白水平：与疾病严重程度的关系。

Arch Dermatol Res. 1994;286(7):420-2. doi: 10.1007/BF00371804.

Elevated serum-soluble ELAM-1 levels in patients with severe plaque-type psoriasis.重度斑块型银屑病患者血清可溶性ELAM-1水平升高。

Arch Dermatol Res. 1994;286(7):414-6. doi: 10.1007/BF00371802.

本文引用的文献

Purified intercellular adhesion molecule-1 (ICAM-1) is a ligand for lymphocyte function-associated antigen 1 (LFA-1).纯化的细胞间黏附分子-1（ICAM-1）是淋巴细胞功能相关抗原1（LFA-1）的配体。

Cell. 1987 Dec 4;51(5):813-9. doi: 10.1016/0092-8674(87)90104-8.

The function of human intercellular adhesion molecule-1 (ICAM-1) in the generation of an immune response.人类细胞间黏附分子-1（ICAM-1）在免疫反应产生中的作用。

Eur J Immunol. 1988 Jan;18(1):35-9. doi: 10.1002/eji.1830180107.

Kinetics and characterization of intercellular adhesion molecule-1 (ICAM-1) expression on keratinocytes in various inflammatory skin lesions and malignant cutaneous lymphomas.不同炎症性皮肤病变和恶性皮肤淋巴瘤中角质形成细胞上细胞间黏附分子-1（ICAM-1）表达的动力学及特征

J Am Acad Dermatol. 1989 May;20(5 Pt 1):782-90. doi: 10.1016/s0190-9622(89)70090-6.

Characterization of intercellular adhesion molecule-1 and HLA-DR expression in normal and inflamed skin: modulation by recombinant gamma interferon and tumor necrosis factor.正常皮肤和炎症皮肤中细胞间黏附分子-1及人类白细胞抗原-DR表达的特征：重组γ干扰素和肿瘤坏死因子的调节作用

J Am Acad Dermatol. 1989 Apr;20(4):617-29. doi: 10.1016/s0190-9622(89)70073-6.

The effect of in vivo interferon-gamma on the distribution of LFA-1 and ICAM-1 in normal human skin.体内干扰素-γ对正常人皮肤中淋巴细胞功能相关抗原-1和细胞间黏附分子-1分布的影响

J Invest Dermatol. 1989 Oct;93(4):439-42. doi: 10.1111/1523-1747.ep12284016.

Intercellular adhesion molecule-I (ICAM-I) expression correlated to inflammation.细胞间黏附分子-1（ICAM-1）的表达与炎症相关。

Br J Dermatol. 1989 Apr;120(4):479-84. doi: 10.1111/j.1365-2133.1989.tb01320.x.

Epidermal keratinocytes express the adhesion molecule intercellular adhesion molecule-1 in inflammatory dermatoses.

J Invest Dermatol. 1989 May;92(5):746-50. doi: 10.1111/1523-1747.ep12722441.

Adhesion molecule expression in psoriatic skin lesions and the influence of cyclosporin A.银屑病皮肤病变中黏附分子的表达及环孢素A的影响。

Clin Exp Immunol. 1991 Apr;84(1):157-62. doi: 10.1111/j.1365-2249.1991.tb08140.x.

ICAM-1 (CD54): a counter-receptor for Mac-1 (CD11b/CD18).细胞间黏附分子-1（CD54）：巨噬细胞-1抗原（CD11b/CD18）的反受体

J Cell Biol. 1990 Dec;111(6 Pt 2):3129-39. doi: 10.1083/jcb.111.6.3129.

UM4D4+ (CDw60) T cells are compartmentalized into psoriatic skin and release lymphokines that induce a keratinocyte phenotype expressed in psoriatic lesions.

J Invest Dermatol. 1990 Sep;95(3):275-82. doi: 10.1111/1523-1747.ep12484908.

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