Cooper R S
Department of Preventive Medicine and Epidemiology, Loyola University Medical School, Maywood, IL 60153.
Ann Epidemiol. 1993 Mar;3(2):137-44. doi: 10.1016/1047-2797(93)90126-o.
Inequalities in health status among various racial and ethnic groups form a prominent chapter in the history of public health in the United States. Inequalities are observed for a wide range of specific conditions, and evolve over time. Little evidence is available to support the contention that any sizable proportion of this overall inequality is caused by genetic factors operating at the population level. On the other hand, social status, as measured by proxy variables like income and education, does provide important information about the black-white differential in health status when used in standard epidemiologic analyses. What is often neglected in these analyses, however, is an attempt to define underlying pathogenic mechanisms that incorporate social processes. Causal explanations based on the broader observation that inequality determined by the structure of social relations is associated with health differentials in all populations offer a plausible theoretic framework. At the same time, the unique character of the experience of specific ethnic subpopulations must be accounted for. Any attempt to account for the impact of race with greater precision will require an attempt to define this demographic variable within the context of social class relations.
美国公共卫生史上,不同种族和族裔群体在健康状况方面的不平等是一个显著的篇章。在一系列具体病症中都存在不平等现象,且会随时间演变。几乎没有证据支持这样的观点,即总体不平等中有相当大比例是由群体层面的遗传因素导致的。另一方面,用收入和教育等替代变量衡量的社会地位,在标准流行病学分析中用于解释健康状况的黑白差异时,确实能提供重要信息。然而,这些分析中常常被忽视的是,试图界定包含社会过程的潜在致病机制。基于更广泛观察的因果解释,即由社会关系结构决定的不平等与所有人群的健康差异相关,提供了一个合理的理论框架。与此同时,必须考虑特定族裔亚群体经历的独特性。任何更精确解释种族影响的尝试,都需要在社会阶级关系背景下界定这一人口统计学变量。