Gurevitch J, Sela B, Jonas A, Golan H, Yahav Y, Passwell J H
Department of Pediatrics, Sheba Medical Center, Tel Hashomer, Israel.
Acta Paediatr. 1993 Jan;82(1):119-21. doi: 10.1111/j.1651-2227.1993.tb12538.x.
A 20-month old infant, who had short bowel syndrome following extensive surgery for a mid gut volvulus, developed hyperchloremic acidosis, with a large anion gap after enteral feeding was instituted. The organic acidosis was at least partly due to an increased concentration of D-lactic acid. This patient, as did five other pediatric patients, presented with a typical encephalopathy syndrome. Early recognition of this syndrome and treatment with an intestinal antibiotic and discontinuation of enteral feeding enabled prompt correction of the hyperchloremic acidosis and a rapid clinical recovery.
一名20个月大的婴儿，因中肠扭转接受广泛手术后出现短肠综合征，在开始肠内喂养后发生高氯性酸中毒，并伴有较大的阴离子间隙。有机酸血症至少部分归因于D-乳酸浓度升高。该患者与其他五名儿科患者一样，出现典型的脑病综合征。早期识别该综合征并使用肠道抗生素治疗以及停止肠内喂养，可使高氯性酸中毒迅速得到纠正，并实现快速临床康复。
