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大肠杆菌O157:H7中rpoS对酸、热和盐耐受性的调控

rpoS regulation of acid, heat, and salt tolerance in Escherichia coli O157:H7.

作者信息

Cheville A M, Arnold K W, Buchrieser C, Cheng C M, Kaspar C W

机构信息

Department of Food Microbiology and Toxicology, University of Wisconsin, Madison 53706-1187, USA.

出版信息

Appl Environ Microbiol. 1996 May;62(5):1822-4. doi: 10.1128/aem.62.5.1822-1824.1996.

Abstract

An rpoS mutant (rpoS::pRR10) of Escherichia coli O157:H7 ATCC 43895 was generated. Stationary-phase acid, heat, and salt tolerance was significantly reduced, and starvation-induced acid tolerance did not develop in the mutant. RpoS was also important for survival of E. coli O157:H7 in dry, fermented sausage.

摘要

构建了大肠杆菌O157:H7 ATCC 43895的rpoS突变体(rpoS::pRR10)。该突变体在稳定期的耐酸性、耐热性和耐盐性显著降低,且不会产生饥饿诱导的耐酸性。RpoS对大肠杆菌O157:H7在干燥发酵香肠中的存活也很重要。

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本文引用的文献

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Induction of Carbon Starvation-Induced Proteins in Vibrio vulnificus.诱导创伤弧菌产生碳饥饿诱导蛋白。
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Acid resistance in enteric bacteria.肠道细菌中的耐酸性
Infect Immun. 1993 Jan;61(1):364-7. doi: 10.1128/iai.61.1.364-367.1993.
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Acid tolerance of enterohemorrhagic Escherichia coli.肠出血性大肠杆菌的耐酸性
Appl Environ Microbiol. 1995 Apr;61(4):1669-72. doi: 10.1128/aem.61.4.1669-1672.1995.

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