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睡眠呼吸暂停、过度嗜睡以及腺样体扁桃体肥大继发的上气道阻塞。

Sleep apnea, hypersomnolence, and upper airway obstruction secondary to adenotonsillar enlargement.

作者信息

Mangat D, Orr W C, Smith R O

出版信息

Arch Otolaryngol. 1977 Jul;103(7):383-6. doi: 10.1001/archotol.1977.00780240041003.

Abstract

The authors examined four children with substantial adenotonsillar enlargement that caused intermittent severe upper airway obstructive episodes, documented by all-night polygraphic monitoring of sleep and respirations. Previously unreported complications of severe adenotonsillar enlargement that were demonstrated included prolonged obstructive apneic episodes and disturbed sleep patterns. The children also exhibited daytime hypersomnolence. Polygraphic sleep studies after adenotonsillectomy confirmed improved sleep patterns and relief of the obstructive episodes. None of our patienqs had developed pulmonary hypertension, cor pulmonale, or other reported complications of prolonged upper airway obstruction, all of which may have been averted by early diagnosis and treatment.

摘要

作者检查了4名患有腺样体扁桃体明显肿大的儿童,这些肿大导致间歇性严重上呼吸道阻塞发作,通过对睡眠和呼吸进行整夜多导睡眠监测得以证实。所证实的严重腺样体扁桃体肿大先前未报告的并发症包括长时间阻塞性呼吸暂停发作和睡眠模式紊乱。这些儿童还表现出日间嗜睡。腺样体扁桃体切除术后的多导睡眠研究证实睡眠模式得到改善,阻塞性发作得到缓解。我们的患者均未发生肺动脉高压、肺心病或其他报告的长时间上呼吸道阻塞并发症,所有这些并发症本可通过早期诊断和治疗得以避免。

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