Sampson H W
Department of Human Anatomy and Neurobiology, College of Medicine, Texas A & M University, Health Science Center, College Station 77843-1114, USA.
Alcohol Clin Exp Res. 1997 May;21(3):400-3. doi: 10.1111/j.1530-0277.1997.tb03782.x.
The mechanism of the production of ethanol-associated osteopenia seems to be a direct effect of alcohol on bone cells and an indirect or modulating effect through mineral regulating hormones such as vitamin D metabolites, parathyroid hormone, and calcitonin. The modulating effects of these hormones on bone and mineral metabolism in acute and chronic alcohol consumption is discussed herein.
乙醇相关性骨质减少的产生机制似乎是酒精对骨细胞的直接作用，以及通过矿物质调节激素（如维生素D代谢产物、甲状旁腺激素和降钙素）产生的间接或调节作用。本文讨论了这些激素在急性和慢性酒精摄入中对骨和矿物质代谢的调节作用。
