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1
Infection of macrophages by Theiler's murine encephalomyelitis virus is highly dependent on their activation or differentiation state.泰勒氏鼠脑脊髓炎病毒对巨噬细胞的感染高度依赖于其激活或分化状态。
J Virol. 1997 Nov;71(11):8864-7. doi: 10.1128/JVI.71.11.8864-8867.1997.
2
Theiler's virus growth in murine macrophage cell lines depends on the state of differentiation.
Virology. 1995 Jun 1;209(2):437-44. doi: 10.1006/viro.1995.1276.
3
The influence of macrophage growth factors on Theiler's Murine Encephalomyelitis Virus (TMEV) infection and activation of macrophages.巨噬细胞生长因子对 Theiler's 鼠脑脊髓炎病毒（TMEV）感染和巨噬细胞激活的影响。
Cytokine. 2018 Feb;102:83-93. doi: 10.1016/j.cyto.2017.07.015. Epub 2017 Aug 9.
4
Theiler's murine encephalomyelitis virus subgroup strain-specific infection in a murine macrophage-like cell line.泰勒氏小鼠脑脊髓炎病毒亚组毒株在小鼠巨噬细胞样细胞系中的特异性感染
J Virol. 1997 Jan;71(1):729-33. doi: 10.1128/JVI.71.1.729-733.1997.
5
Distinct cell death mechanisms by Theiler's murine encephalomyelitis virus (TMEV) infection in microglia and macrophage.微小胶质细胞和巨噬细胞中泰勒氏鼠脑脊髓炎病毒（TMEV）感染导致的不同细胞死亡机制
Neurosci Lett. 2002 Jul 12;327(1):41-4. doi: 10.1016/s0304-3940(02)00376-2.
6
Two models of multiple sclerosis: experimental allergic encephalomyelitis (EAE) and Theiler's murine encephalomyelitis virus (TMEV) infection. A pathological and immunological comparison.多发性硬化的两种模型：实验性变应性脑脊髓炎（EAE）和泰勒鼠脑脊髓炎病毒（TMEV）感染。病理与免疫学比较。
Microsc Res Tech. 1995 Oct 15;32(3):215-29. doi: 10.1002/jemt.1070320305.
7
A lentiviral expression system demonstrates that L* protein of Theiler's murine encephalomyelitis virus (TMEV) is essential for virus growth in a murine macrophage-like cell line.一种慢病毒表达系统表明，泰勒氏小鼠脑脊髓炎病毒（TMEV）的L*蛋白对于该病毒在小鼠巨噬细胞样细胞系中的生长至关重要。
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8
Control of early Theiler's murine encephalomyelitis virus replication in macrophages by interleukin-6 occurs in conjunction with STAT1 activation and nitric oxide production.白细胞介素-6 通过激活 STAT1 和产生一氧化氮来控制巨噬细胞中早期脊髓灰质炎病毒的复制。
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Astrocytes, not microglia, are the main cells responsible for viral persistence in Theiler's murine encephalomyelitis virus infection leading to demyelination.在导致脱髓鞘的泰勒氏鼠脑脊髓炎病毒感染中，星形胶质细胞而非小胶质细胞是负责病毒持续存在的主要细胞。
J Neuroimmunol. 2001 Aug 30;118(2):256-67. doi: 10.1016/s0165-5728(01)00338-1.
10
Persistent infection of RAW264.7 macrophages with the DA strain of Theiler's murine encephalomyelitis virus: An in vitro model to study viral persistence.用泰勒氏鼠脑脊髓炎病毒DA株持续感染RAW264.7巨噬细胞：一种研究病毒持续性感染的体外模型。
J Neurovirol. 2006 Apr;12(2):108-15. doi: 10.1080/13550280600714120.

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1
The Saffold Virus-Penang 2B and 3C Proteins, but not the L Protein, Induce Apoptosis in HEp-2 and Vero Cells.萨夫登病毒-槟城 2B 和 3C 蛋白，但不是 L 蛋白，诱导 HEp-2 和 Vero 细胞凋亡。
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Modeling the acute and chronic phases of Theiler murine encephalomyelitis virus infection.建立东方田鼠脑脊髓炎病毒感染的急、慢性期模型。
J Virol. 2013 Apr;87(7):4052-9. doi: 10.1128/JVI.03395-12. Epub 2013 Jan 30.
4
Modulation of viral replication in macrophages persistently infected with the DA strain of Theiler's murine encephalomyelitis virus.持续感染泰勒氏小鼠脑脊髓炎病毒DA株的巨噬细胞中病毒复制的调节
Virol J. 2008 Aug 4;5:89. doi: 10.1186/1743-422X-5-89.
5
Apoptotic cells, including macrophages, are prominent in Theiler's virus-induced inflammatory, demyelinating lesions.凋亡细胞，包括巨噬细胞，在泰勒氏病毒诱导的炎症性脱髓鞘病变中很突出。
J Virol. 2003 Apr;77(7):4383-8. doi: 10.1128/jvi.77.7.4383-4388.2003.
6
Theiler's virus infection of primary cultures of bone marrow-derived monocytes/macrophages.骨髓来源的单核细胞/巨噬细胞原代培养物的泰勒氏病毒感染
J Virol. 2002 Dec;76(24):12823-33. doi: 10.1128/jvi.76.24.12823-12833.2002.
7
Non-AUG-initiated internal translation of the L* protein of Theiler's virus and importance of this protein for viral persistence.泰勒氏病毒L*蛋白的非AUG起始内部翻译及其对病毒持续性的重要性。
J Virol. 2002 Nov;76(21):10665-73. doi: 10.1128/jvi.76.21.10665-10673.2002.
8
High numbers of viral RNA copies in the central nervous system of mice during persistent infection with Theiler's virus.在感染泰勒氏病毒的小鼠持续感染期间，其中枢神经系统中存在大量病毒RNA拷贝。
J Virol. 2001 Aug;75(16):7420-8. doi: 10.1128/JVI.75.16.7420-7428.2001.
9
Influence of the Theiler's virus L* protein on macrophage infection, viral persistence, and neurovirulence.泰勒氏病毒L*蛋白对巨噬细胞感染、病毒持续性及神经毒力的影响。
J Virol. 2000 Oct;74(19):9071-7. doi: 10.1128/jvi.74.19.9071-9077.2000.
10
Analysis of cellular mutants resistant to Theiler's virus infection: differential infection of L929 cells by persistent and neurovirulent strains.对抵抗泰勒氏病毒感染的细胞突变体的分析：持久性毒株和神经毒力毒株对L929细胞的差异感染
J Virol. 1999 Sep;73(9):7248-54. doi: 10.1128/JVI.73.9.7248-7254.1999.

本文引用的文献

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Observations on encephalomyelitis of mice (DA strain).对小鼠（DA品系）脑脊髓炎的观察
J Exp Med. 1952 Dec;96(6):517-30. doi: 10.1084/jem.96.6.517.
2
Role of macrophages during Theiler's virus infection.巨噬细胞在泰勒氏病毒感染中的作用。
J Virol. 1997 Apr;71(4):3336-40. doi: 10.1128/JVI.71.4.3336-3340.1997.
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Theiler's murine encephalomyelitis virus subgroup strain-specific infection in a murine macrophage-like cell line.泰勒氏小鼠脑脊髓炎病毒亚组毒株在小鼠巨噬细胞样细胞系中的特异性感染
J Virol. 1997 Jan;71(1):729-33. doi: 10.1128/JVI.71.1.729-733.1997.
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The predominant virus antigen burden is present in macrophages in Theiler's murine encephalomyelitis virus-induced demyelinating disease.在泰勒氏鼠脑脊髓炎病毒诱导的脱髓鞘疾病中，主要的病毒抗原负荷存在于巨噬细胞中。
J Virol. 1995 Apr;69(4):2525-33. doi: 10.1128/JVI.69.4.2525-2533.1995.
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Theiler's virus growth in murine macrophage cell lines depends on the state of differentiation.
Virology. 1995 Jun 1;209(2):437-44. doi: 10.1006/viro.1995.1276.
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Theiler's virus persists in glial cells during demyelinating disease.泰勒氏病毒在脱髓鞘疾病期间持续存在于神经胶质细胞中。
Cell. 1981 Oct;26(1 Pt 1):123-8. doi: 10.1016/0092-8674(81)90040-4.
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A highly sensitive cell line, WEHI 164 clone 13, for measuring cytotoxic factor/tumor necrosis factor from human monocytes.一种用于检测人单核细胞细胞毒性因子/肿瘤坏死因子的高敏感性细胞系——WEHI 164克隆13。
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Internal initiation of translation of eukaryotic mRNA directed by a sequence derived from poliovirus RNA.由源自脊髓灰质炎病毒RNA的序列指导的真核生物mRNA的内部翻译起始。
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Molecular cloning of the complete genome of strain GDVII of Theiler's virus and production of infectious transcripts.泰勒氏病毒GDVII株全基因组的分子克隆及感染性转录本的产生。
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The methylation-free status of a housekeeping transgene is lost at high copy number.持家转基因的无甲基化状态在高拷贝数时会丧失。
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泰勒氏鼠脑脊髓炎病毒对巨噬细胞的感染高度依赖于其激活或分化状态。

Infection of macrophages by Theiler's murine encephalomyelitis virus is highly dependent on their activation or differentiation state.

作者信息

Shaw-Jackson C, Michiels T

机构信息

International Institute of Cellular and Molecular Pathology, University of Louvain, Brussels, Belgium.

出版信息

J Virol. 1997 Nov;71(11):8864-7. doi: 10.1128/JVI.71.11.8864-8867.1997.

DOI:10.1128/JVI.71.11.8864-8867.1997
PMID:9343249
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC192355/
Abstract

Macrophages are the main targets of Theiler's murine encephalomyelitis virus (TMEV) during persistent demyelinating infection of mice. Replication of TMEV in macrophages was previously shown to depend on their activation state. Here, we show that the quality of the serum used for culture drastically influences viral entry in RAW264.7 macrophages.

摘要

巨噬细胞是小鼠在持续脱髓鞘感染期间泰勒氏鼠脑脊髓炎病毒（TMEV）的主要靶标。先前已表明TMEV在巨噬细胞中的复制取决于它们的激活状态。在这里，我们表明用于培养的血清质量会极大地影响RAW264.7巨噬细胞中的病毒进入。