Malagelada J R, Holtermuller K H, Sizemore G W, Go V L
Gastroenterology. 1976 Sep;71(3):405-8.
Hypercalcemia produced in normal volunteers by intravenous infusions of CaCl2 increased outputs of pancreatic enzyme and gastric acid. Further, hypercalcemia enhanced cholecystokinin (CCK)-stimulated pancreatic enzyme secretion and gallbladder contraction. Our results suggest that hypercalcemia affects pancreatic, gallbladder, and gastric functions as well as their responses to exogenous CCK.
通过静脉输注氯化钙在正常志愿者中产生的高钙血症增加了胰酶和胃酸的分泌量。此外,高钙血症增强了胆囊收缩素(CCK)刺激的胰酶分泌和胆囊收缩。我们的结果表明,高钙血症会影响胰腺、胆囊和胃的功能以及它们对外源性CCK的反应。