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Purification and properties of a membrane-bound lytic transglycosylase from Escherichia coli.大肠杆菌膜结合溶菌转糖基酶的纯化及性质
J Bacteriol. 1994 Jan;176(2):338-43. doi: 10.1128/jb.176.2.338-343.1994.
2
Beta-lactam-induced bacteriolysis of amino acid-deprived Escherichia coli is dependent on phospholipid synthesis.β-内酰胺诱导的氨基酸缺乏型大肠杆菌的细菌溶解依赖于磷脂合成。
J Bacteriol. 1995 Feb;177(4):992-7. doi: 10.1128/jb.177.4.992-997.1995.
3
Direct correlation between overproduction of guanosine 3',5'-bispyrophosphate (ppGpp) and penicillin tolerance in Escherichia coli.大肠杆菌中鸟苷 3',5'-双焦磷酸（ppGpp）过量产生与青霉素耐受性之间的直接关联。
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On the nature of the control by RC gene in e. coli: amino acid-dependent control of lipid synthesis.关于大肠杆菌中RC基因的控制性质：脂质合成的氨基酸依赖性控制。
Biochem Biophys Res Commun. 1968 Oct 10;33(1):108-12. doi: 10.1016/0006-291x(68)90263-5.
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Multiple antibiotic resistance in a bacterium with suppressed autolytic system.具有受抑制自溶系统的细菌中的多重抗生素耐药性
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Temperature sensitivity of the penicillin-induced autolysis mechanism in nongrowing cultures of Escherichia coli.大肠杆菌非生长培养物中青霉素诱导的自溶机制的温度敏感性
J Bacteriol. 1987 May;169(5):2310-2. doi: 10.1128/jb.169.5.2310-2312.1987.
7
Dissociation of the ampicillin-induced lysis of amino acid-deprived Escherichia coli into two stages.氨苄青霉素诱导的氨基酸缺乏型大肠杆菌裂解分为两个阶段。
J Bacteriol. 1990 Apr;172(4):2187-90. doi: 10.1128/jb.172.4.2187-2190.1990.
8
Lysis of Escherichia coli by beta-lactams which bind penicillin-binding proteins 1a and 1b: inhibition by heat shock proteins.与青霉素结合蛋白1a和1b结合的β-内酰胺对大肠杆菌的裂解作用：热休克蛋白的抑制作用
J Bacteriol. 1991 Jul;173(13):4021-6. doi: 10.1128/jb.173.13.4021-4026.1991.
9
Stringent control of peptidoglycan biosynthesis in Escherichia coli K-12.大肠杆菌K-12中肽聚糖生物合成的严格控制。
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β-内酰胺类抗生素在氨基酸缺乏的大肠杆菌中诱导细菌裂解的温度敏感性

Temperature sensitivity of bacteriolysis induced by beta-lactam antibiotics in amino acid-deprived Escherichia coli.

作者信息

Rodionov D G, Ishiguro E E

机构信息

Department of Biochemistry and Microbiology, University of Victoria, British Columbia, Canada.

出版信息

J Bacteriol. 1998 Apr;180(8):2224-7. doi: 10.1128/JB.180.8.2224-2227.1998.

DOI:10.1128/JB.180.8.2224-2227.1998
PMID:9555908
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC107152/
Abstract

The temperature-sensitive penicillin tolerance response previously reported in amino acid-deprived Escherichia coli (W. Kusser and E. E. Ishiguro, J. Bacteriol. 169:2310-2312, 1987) was not due to the induction of the heat shock response resulting from a temperature upshift and was therefore unrelated to the findings of another report (J. K. Powell and K. D. Young, J. Bacteriol. 173:4021-4026, 1991) indicating a positive correlation between the expression of heat shock proteins and penicillin tolerance. The thermosensitive event occurred in the lysis induction stage.

摘要

先前在氨基酸缺乏的大肠杆菌中报道的温度敏感型青霉素耐受反应（W. 库塞尔和E. E. 石黑，《细菌学杂志》169:2310 - 2312，1987年）并非由于温度升高引发热休克反应所致，因此与另一篇报告（J. K. 鲍威尔和K. D. 杨，《细菌学杂志》173:4021 - 4026，1991年）的研究结果无关，该报告指出热休克蛋白的表达与青霉素耐受性之间存在正相关。热敏事件发生在裂解诱导阶段。