慢性活动性EB病毒感染伴基底节钙化。
Calcification in the basal ganglia with chronic active Epstein-Barr virus infection.
作者信息
Morita M, Tsuge I, Matsuoka H, Ito Y, Itosu T, Yamamoto M, Morishima T
机构信息
Department of Pediatrics, Nagoya University School of Medicine, Japan.
出版信息
Neurology. 1998 May;50(5):1485-8. doi: 10.1212/wnl.50.5.1485.
PMID:9596016
Abstract
We report three Japanese children with chronic active Epstein-Barr virus (EBV) infection and neurologic findings that included symmetric calcifications in the bilateral basal ganglia. This finding, common in pediatric acquired immunodeficiency syndrome (AIDS), suggests that EBV could be responsible for calcification in the basal ganglia seen in children with AIDS and in other idiopathic diseases.
摘要
我们报告了三名患有慢性活动性爱泼斯坦-巴尔病毒（EBV）感染且有神经系统表现的日本儿童，这些表现包括双侧基底神经节对称性钙化。这一在儿童获得性免疫缺陷综合征（AIDS）中常见的发现表明，EBV可能是导致AIDS患儿以及其他特发性疾病患儿基底神经节钙化的原因。
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