四氯化碳在内质网中产生的变化。 - Suppr

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Alterations produced in the endoplasmic reticulum by carbon tetrachloride.

作者信息

Smuckler E A

出版信息

Panminerva Med. 1976 Sep-Oct;18(9-10):292-309.

PMID:980529

Abstract

摘要

相似文献

Alterations produced in the endoplasmic reticulum by carbon tetrachloride.四氯化碳在内质网中产生的变化。

Panminerva Med. 1976 Sep-Oct;18(9-10):292-309.

Differences in the carbon tetrachloride-induced damage to components of the smooth and rough endoplasmic reticulum from rat liver.

Biochem Biophys Res Commun. 1973 Jan 23;50(2):337-43. doi: 10.1016/0006-291x(73)90845-0.

Liver parenchymal cell injury. IX. Phenobarbital potentiation of endoplasmic reticulum denaturation following carbon tetrachloride.肝实质细胞损伤。IX. 四氯化碳后内质网变性的苯巴比妥增强作用。

Lab Invest. 1972 Mar;26(3):290-9.

Liver parenchymal cell injury. VII. Membrane denaturation following carbon tetrachloride.肝实质细胞损伤。VII. 四氯化碳诱导的膜变性

Lab Invest. 1971 Sep;25(3):269-78.

Interference of CCI4 metabolites with subcellular structures.四氯化碳代谢产物对亚细胞结构的干扰。

Panminerva Med. 1976 Sep-Oct;18(9-10):310-9.

[Ultrastructural changes in parenchymatous liver cell during carbon tetrachloride poisoning of intact and adrenalectomized rats].[完整及肾上腺切除大鼠四氯化碳中毒时实质肝细胞的超微结构变化]

Tsitologiia. 1971 Apr;13(4):441-6.

[Changes in the thyroid gland and adrenals in prolonged carbon tetrachloride poisoning].[长期四氯化碳中毒时甲状腺和肾上腺的变化]

Biull Eksp Biol Med. 1970 Sep;70(9):109-13.

[Atypical mitosis in liver cells in experimental hepatitis and cirrhosis].[实验性肝炎和肝硬化中肝细胞的非典型有丝分裂]

Arkh Patol. 1971;33(11):62-6.

Characteristic lesions of methylazoxymethanol-induced liver damage. A comparative ultrastructural study with dimethylnitrosamine, hydrazine sulfate, and carbon tetrachloride.

Lab Invest. 1968 Oct;19(4):382-98.

[Research on the importance of investigating lymph node reactions in cirrhogenic hepatopathy].[关于研究肝硬化性肝病中淋巴结反应重要性的研究]

Rev Med Interna Neurol Psihiatr Neurochir Dermatovenerol Med Interna. 1977 Mar-Apr;29(2):157-61.

引用本文的文献

Decreased incorporation of 14C-leucine in different liver nuclear protein fractions at early stages of carbon tetrachloride poisoning in the rat.在大鼠四氯化碳中毒早期，不同肝细胞核蛋白组分中14C-亮氨酸的掺入减少。

Arch Toxicol. 1994;68(3):206-9. doi: 10.1007/s002040050056.

Potential for free radical-induced lipid peroxidation as a cause of endothelial cell injury in Rocky Mountain spotted fever.自由基诱导的脂质过氧化作为落基山斑疹热中内皮细胞损伤原因的可能性。

Infect Immun. 1988 Dec;56(12):3110-5. doi: 10.1128/iai.56.12.3110-3115.1988.

Early hypomethylation of 2'-O-ribose moieties in hepatocyte cytoplasmic ribosomal RNA underlies the protein synthetic defect produced by CCl4.四氯化碳导致的蛋白质合成缺陷的基础是肝细胞胞质核糖体核糖核酸中2'-O-核糖部分的早期低甲基化。

J Cell Biol. 1987 Aug;105(2):705-11. doi: 10.1083/jcb.105.2.705.

Changes in serum proteins and in major liver constituents following carbon tetrachloride and a high-fat choline-free diet in rats.

Experientia. 1979 Sep 15;35(9):1225-6. doi: 10.1007/BF01963304.

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