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Fatty acid ethyl esters: ethanol metabolites which mediate ethanol-induced organ damage and serve as markers of ethanol intake.

作者信息

Laposata M

机构信息

Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston, USA.

出版信息

Prog Lipid Res. 1998 Nov;37(5):307-16. doi: 10.1016/s0163-7827(98)00013-7.

DOI:10.1016/s0163-7827(98)00013-7
PMID:10209651
Abstract
摘要

相似文献

1
Fatty acid ethyl esters: ethanol metabolites which mediate ethanol-induced organ damage and serve as markers of ethanol intake.
Prog Lipid Res. 1998 Nov;37(5):307-16. doi: 10.1016/s0163-7827(98)00013-7.
2
Fatty acid ethyl esters: toxic non-oxidative metabolites of ethanol and markers of ethanol intake.
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Fatty acid ethyl esters: short-term and long-term serum markers of ethanol intake.脂肪酸乙酯:乙醇摄入的短期和长期血清标志物。
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4
Fatty acid ethyl esters: nonoxidative ethanol metabolites with emerging biological and clinical significance.脂肪酸乙酯:具有新出现的生物学和临床意义的非氧化乙醇代谢产物。
Lipids. 1999;34 Suppl:S281-5. doi: 10.1007/BF02562318.
5
Presence of nonoxidative ethanol metabolism in human organs commonly damaged by ethanol abuse.在通常因酒精滥用而受损的人体器官中存在非氧化乙醇代谢。
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6
Metabolism of ethanol by human brain to fatty acid ethyl esters.人脑将乙醇代谢为脂肪酸乙酯。
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Effects of L-carnitine on the formation of fatty acid ethyl esters in brain and peripheral organs after short-term ethanol administration in rat.
Neurochem Res. 1999 Jan;24(1):79-84. doi: 10.1023/a:1020984114824.
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Nonoxidative ethanol metabolism: formation of fatty acid ethyl esters by cholesterol esterase.非氧化乙醇代谢:胆固醇酯酶催化脂肪酸乙酯的形成。
Proc Natl Acad Sci U S A. 1982 Jul;79(13):3954-7. doi: 10.1073/pnas.79.13.3954.
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Fatty acid ethyl esters: potentially toxic products of myocardial ethanol metabolism.
J Mol Cell Cardiol. 1998 Nov;30(11):2487-94. doi: 10.1006/jmcc.1998.0812.
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Fatty acid ethyl esters: current facts and speculations.脂肪酸乙酯:当前的事实与推测
Prostaglandins Leukot Essent Fatty Acids. 1999 May-Jun;60(5-6):313-5. doi: 10.1016/s0952-3278(99)80005-2.

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Fatty acid ethyl esters disrupt neonatal alveolar macrophage mitochondria and derange cellular functioning.脂肪酸乙酯会破坏新生儿肺泡巨噬细胞的线粒体并扰乱细胞功能。
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Chronic ethanol consumption increases cardiomyocyte fatty acid uptake and decreases ventricular contractile function in C57BL/6J mice.慢性乙醇摄入增加 C57BL/6J 小鼠心肌细胞脂肪酸摄取,降低心室收缩功能。
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Ethyl arachidonate is the predominant fatty acid ethyl ester in the brains of alcohol-intoxicated subjects at autopsy.花生四烯酸乙酯是尸检时酒精中毒者大脑中主要的脂肪酸乙酯。
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