胎膜早破、先兆子痫和呼吸窘迫综合征。
Prolonged rupture of membranes, pre-eclamptic toxaemia, and respiratory distress syndrome.
作者信息
Chiswick M L
出版信息
Arch Dis Child. 1976 Sep;51(9):674-9. doi: 10.1136/adc.51.9.674.
Abstract
A retrospective analysis was made of the incidence and severity of the idiopathic respiratory distress syndrome (RDS) in babies of less than 35 weeks' gestation born at this hospital from January 1967-December 1974. There was a lower incidence of RDS in babies born after pregnancies complicated only by prolonged rupture of membranes (PRM) (19%) and in babies born vaginally after pregnancies complicated only by pre-eclamptic toxaemia (PET) (18%) compared with the incidence of RDS after uncomplicated pregnancies (35%). Babies born vaginally who developed RDS after pregnancies complicated by PRM or PET had less severe disease compared with those who developed RDS after uncomplicated pregnancies. Mortality in babies who developed severe RDS was not influenced by the occurrence of PRM or PET. The biological implication of the study is that certain complications of pregnancy may accelerate pulmonary surfactant production in preterm babies.
摘要
对1967年1月至1974年12月在本院出生的孕周小于35周的婴儿特发性呼吸窘迫综合征（RDS）的发病率和严重程度进行了回顾性分析。与无并发症妊娠后RDS的发病率（35%）相比，仅因胎膜早破（PRM）而并发妊娠的婴儿中RDS的发病率较低（19%），以及仅因先兆子痫（PET）而并发妊娠后经阴道分娩的婴儿中RDS的发病率较低（18%）。与无并发症妊娠后发生RDS的婴儿相比，因PRM或PET而并发妊娠后经阴道分娩且发生RDS的婴儿病情较轻。发生严重RDS的婴儿的死亡率不受PRM或PET的影响。该研究的生物学意义在于，妊娠的某些并发症可能会加速早产儿肺表面活性物质的产生。
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