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幼儿有样学样?基因与环境对实验室评估的诱发模仿的影响

Toddler see, toddler do? Genetic and environmental influences on laboratory-assessed elicited imitation.

作者信息

Fenstermacher Susan K, Saudino Kimberly J

机构信息

Department of Psychology, Boston University, 64 Cummington Street, Boston, MA 02215, USA.

出版信息

Behav Genet. 2007 Sep;37(5):639-47. doi: 10.1007/s10519-007-9160-5. Epub 2007 Jun 14.

DOI:10.1007/s10519-007-9160-5
PMID:17566859
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4108219/
Abstract

The imitative performance of 311 pairs of 24-month old twins (143 MZ, 168 same-sex DZ) was assessed via three multi-step imitative sequences. Composite imitation score correlations suggested the presence of genetic influences on imitation, with MZ correlations significantly exceeding DZ correlations. Univariate model-fitting procedures supported this finding. Substantial broad heritability was found for imitative performance, with no evidence for shared environment. However, we are unable to say with certainty to what extent this heritability is represented by additive and nonadditive genetic variance. Estimates of heritability derived from both ACE and ADE model-fitting procedures accounted for approximately 50% of the total variance, with the remaining variance in imitative performance attributable to nonshared environmental factors.

摘要

通过三个多步骤模仿序列对311对24个月大的双胞胎(143对同卵双胞胎,168对同性异卵双胞胎)的模仿表现进行了评估。综合模仿分数相关性表明存在对模仿的遗传影响,同卵双胞胎的相关性显著超过异卵双胞胎的相关性。单变量模型拟合程序支持了这一发现。发现模仿表现具有显著的广义遗传力,没有共享环境的证据。然而,我们无法确定这种遗传力在多大程度上由加性和非加性遗传方差表示。从ACE和ADE模型拟合程序得出的遗传力估计约占总方差的50%,模仿表现的其余方差归因于非共享环境因素。