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1
Stem cells tropism for malignant gliomas.干细胞对恶性胶质瘤的趋向性。
Neurosci Bull. 2007 Nov;23(6):363-9. doi: 10.1007/s12264-007-0054-6.
2
Glioma-produced extracellular matrix influences brain tumor tropism of human neural stem cells.胶质瘤产生的细胞外基质影响人类神经干细胞的脑肿瘤嗜性。
J Neurooncol. 2006 Sep;79(2):125-33. doi: 10.1007/s11060-006-9121-5. Epub 2006 Apr 6.
3
Stem cell-mediated delivery of therapies in the treatment of glioma.干细胞介导的疗法递送在治疗神经胶质瘤中的应用。
Mini Rev Med Chem. 2011 Jun;11(7):591-8. doi: 10.2174/138955711795906897.
4
Neural stem cell tropism to glioma: critical role of tumor hypoxia.神经干细胞对胶质瘤的趋向性：肿瘤缺氧的关键作用。
Mol Cancer Res. 2008 Dec;6(12):1819-29. doi: 10.1158/1541-7786.MCR-08-0146.
5
Novel treatment strategies for malignant gliomas using neural stem cells.使用神经干细胞治疗恶性胶质瘤的新策略。
Neurotherapeutics. 2009 Jul;6(3):458-64. doi: 10.1016/j.nurt.2009.05.003.
6
The tropism of embryoid body cells for glioma cells.
Neuroreport. 2007 Nov 19;18(17):1821-5. doi: 10.1097/WNR.0b013e3282f13e30.
7
In vivo tracking of the tropism of mesenchymal stem cells to malignant gliomas using reporter gene-based MR imaging.利用基于报告基因的磁共振成像对间充质干细胞向恶性脑胶质瘤的趋向性进行体内追踪。
Int J Cancer. 2018 Mar 1;142(5):1033-1046. doi: 10.1002/ijc.31113. Epub 2017 Oct 31.
8
Ionizing radiation augments glioma tropism of mesenchymal stem cells.电离辐射增强间充质干细胞对神经胶质瘤的趋向性。
J Neurosurg. 2018 Jan;128(1):287-295. doi: 10.3171/2016.9.JNS16278. Epub 2017 Mar 31.
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In vivo tracking of superparamagnetic iron oxide nanoparticle-labeled mesenchymal stem cell tropism to malignant gliomas using magnetic resonance imaging. Laboratory investigation.利用磁共振成像对超顺磁性氧化铁纳米颗粒标记的间充质干细胞向恶性胶质瘤的体内趋向性进行追踪。实验室研究。
J Neurosurg. 2008 Feb;108(2):320-9. doi: 10.3171/JNS/2008/108/2/0320.
10
Potent tumor tropism of induced pluripotent stem cells and induced pluripotent stem cell-derived neural stem cells in the mouse intracerebral glioma model.诱导多能干细胞及其衍生的神经干细胞在小鼠脑胶质瘤模型中的强肿瘤趋向性。
Int J Oncol. 2015 Jan;46(1):147-52. doi: 10.3892/ijo.2014.2702. Epub 2014 Oct 8.
引用本文的文献
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To Explore the Stem Cells Homing to GBM: The Rise to the Occasion.探索归巢至胶质母细胞瘤的干细胞：应时而起。
Biomedicines. 2022 Apr 24;10(5):986. doi: 10.3390/biomedicines10050986.
2
Combination treatment with VPA and MSCs‑TRAIL could increase anti‑tumor effects against intracranial glioma.联合应用 VPA 和 MSC-TRAIL 可增强颅内神经胶质瘤的抗肿瘤作用。
Oncol Rep. 2021 Mar;45(3):869-878. doi: 10.3892/or.2021.7937. Epub 2021 Jan 14.
3
Diffuse Intrinsic Pontine Gliomas Exhibit Cell Biological and Molecular Signatures of Fetal Hindbrain-Derived Neural Progenitor Cells.弥漫性内生脑桥胶质瘤具有胎儿后脑来源神经祖细胞的细胞生物学和分子特征。
Neurosci Bull. 2019 Apr;35(2):216-224. doi: 10.1007/s12264-018-00329-6. Epub 2019 Jan 3.
4
Knockdown of SOX2OT inhibits the malignant biological behaviors of glioblastoma stem cells via up-regulating the expression of miR-194-5p and miR-122.敲低 SOX2OT 通过上调 miR-194-5p 和 miR-122 的表达抑制脑胶质瘤干细胞的恶性生物学行为。
Mol Cancer. 2017 Nov 13;16(1):171. doi: 10.1186/s12943-017-0737-1.
5
The Unwanted Cell Migration in the Brain: Glioma Metastasis.大脑中不必要的细胞迁移：胶质瘤转移
Neurochem Res. 2017 Jun;42(6):1847-1863. doi: 10.1007/s11064-017-2272-2. Epub 2017 May 6.
6
Alternative new mesenchymal stem cell source exerts tumor tropism through ALCAM and N-cadherin via regulation of microRNA-192 and -218.替代性新间充质干细胞来源通过ALCAM和N-钙黏蛋白，经由对微小RNA-192和-218的调控发挥肿瘤趋向性。
Mol Cell Biochem. 2017 Mar;427(1-2):177-185. doi: 10.1007/s11010-016-2909-5. Epub 2016 Dec 30.
7
Mesenchymal stem cells from human fat engineered to secrete BMP4 are nononcogenic, suppress brain cancer, and prolong survival.经基因工程改造后分泌骨形态发生蛋白4的人脂肪间充质干细胞无致癌性，可抑制脑癌并延长生存期。
Clin Cancer Res. 2014 May 1;20(9):2375-87. doi: 10.1158/1078-0432.CCR-13-1415.
8
The potential for genetically altered microglia to influence glioma treatment.基因修饰小胶质细胞影响胶质瘤治疗的潜力。
CNS Neurol Disord Drug Targets. 2013 Sep;12(6):750-62. doi: 10.2174/18715273113126660171.
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Mesenchymal stem cells derived from adipose tissue vs bone marrow: in vitro comparison of their tropism towards gliomas.脂肪组织来源的间充质干细胞与骨髓来源的间充质干细胞：体外比较其向神经胶质瘤的趋向性。
PLoS One. 2013;8(3):e58198. doi: 10.1371/journal.pone.0058198. Epub 2013 Mar 12.
10
Neurotransplantation: lux et veritas, fiction or reality?神经移植：光明与真理，虚构还是现实？
J Neurosurg Sci. 2011 Dec;55(4):297-304.
本文引用的文献
1
Malignant gliomas actively recruit bone marrow stromal cells by secreting angiogenic cytokines.恶性胶质瘤通过分泌血管生成细胞因子来积极招募骨髓基质细胞。
J Neurooncol. 2007 Jul;83(3):241-7. doi: 10.1007/s11060-007-9332-4. Epub 2007 Feb 14.
2
Neuronal and glioma-derived stem cell factor induces angiogenesis within the brain.神经元和胶质瘤衍生的干细胞因子可诱导脑内血管生成。
Cancer Cell. 2006 Apr;9(4):287-300. doi: 10.1016/j.ccr.2006.03.003.
3
Glioma-produced extracellular matrix influences brain tumor tropism of human neural stem cells.胶质瘤产生的细胞外基质影响人类神经干细胞的脑肿瘤嗜性。
J Neurooncol. 2006 Sep;79(2):125-33. doi: 10.1007/s11060-006-9121-5. Epub 2006 Apr 6.
4
Vascular endothelial growth factor A contributes to glioma-induced migration of human marrow stromal cells (hMSC).血管内皮生长因子A促进胶质瘤诱导的人骨髓基质细胞（hMSC）迁移。
Exp Neurol. 2006 Jun;199(2):301-10. doi: 10.1016/j.expneurol.2005.11.027. Epub 2006 Mar 29.
5
Chemokines regulate the migration of neural progenitors to sites of neuroinflammation.趋化因子调节神经祖细胞向神经炎症部位的迁移。
J Neurosci. 2006 Mar 22;26(12):3182-91. doi: 10.1523/JNEUROSCI.0156-06.2006.
6
PEX-producing human neural stem cells inhibit tumor growth in a mouse glioma model.产生PEX的人神经干细胞在小鼠胶质瘤模型中抑制肿瘤生长。
Clin Cancer Res. 2005 Aug 15;11(16):5965-70. doi: 10.1158/1078-0432.CCR-05-0371.
7
Brain tumor tropism of transplanted human neural stem cells is induced by vascular endothelial growth factor.血管内皮生长因子诱导移植的人神经干细胞向脑肿瘤的趋向性。
Neoplasia. 2005 Jun;7(6):623-9. doi: 10.1593/neo.04781.
8
Lessons from the bone marrow: how malignant glioma cells attract adult haematopoietic progenitor cells.来自骨髓的启示：恶性胶质瘤细胞如何吸引成年造血祖细胞。
Brain. 2005 Sep;128(Pt 9):2200-11. doi: 10.1093/brain/awh563. Epub 2005 Jun 9.
9
Plasticity and therapeutic potential of mesenchymal stem cells in the nervous system.间充质干细胞在神经系统中的可塑性及治疗潜力
Curr Pharm Des. 2005;11(10):1255-65. doi: 10.2174/1381612053507495.
10
The chemokine stromal cell-derived factor-1 regulates the migration of sensory neuron progenitors.趋化因子基质细胞衍生因子-1调节感觉神经元祖细胞的迁移。
J Neurosci. 2005 Apr 20;25(16):3995-4003. doi: 10.1523/JNEUROSCI.4631-04.2005.
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