Brinkmann Kerstin, Gendolla Guido H E
Department of Psychology, University of Geneva, Switzerland.
J Pers Soc Psychol. 2008 Jan;94(1):146-57. doi: 10.1037/0022-3514.94.1.146.
On the basis of predictions of the mood-behavior model (G. H. E. Gendolla, 2000) and motivational intensity theory (J. W. Brehm & E. A. Self, 1989), the authors conducted 2 studies that critically tested the common assumption that dysphoria is associated with a motivational deficit. Dysphoric and nondysphoric undergraduates performed a cognitive task that was either easy or difficult. Effort intensity (i.e., resource mobilization) was assessed as performance-related cardiovascular reactivity. In support of the authors' predictions and in contrast to the popular view of a general motivational deficit, both studies found a crossover interaction between dysphoria and task difficulty: In the difficult condition, nondysphoric participants indeed showed stronger systolic blood pressure reactivity than dysphoric participants. But in the easy condition, dysphoric participants showed stronger systolic reactivity than nondysphoric participants. The findings are discussed with respect to motivational deficits in depression and possible underlying mechanisms.
基于情绪 - 行为模型(G. H. E. 根多拉,2000年)和动机强度理论(J. W. 布雷姆和E. A. 塞尔夫,1989年)的预测,作者进行了两项研究,对烦躁情绪与动机缺陷相关这一普遍假设进行了严格检验。烦躁不安和无烦躁情绪的大学生完成了一项简单或困难的认知任务。努力强度(即资源调动)被评估为与表现相关的心血管反应性。为支持作者的预测,与普遍认为的一般动机缺陷观点相反,两项研究均发现烦躁情绪与任务难度之间存在交叉交互作用:在困难条件下,无烦躁情绪的参与者确实比烦躁情绪的参与者表现出更强的收缩压反应性。但在简单条件下,烦躁情绪的参与者比无烦躁情绪的参与者表现出更强的收缩反应性。研究结果围绕抑郁症中的动机缺陷及可能的潜在机制展开了讨论。