Clougherty Jane E, Wright Rosalind J, Baxter Lisa K, Levy Jonathan I
Harvard School of Public Health, Department of Environmental Health, Landmark Center 4th Floor West, PO Box 15677, Boston, MA 02215, USA.
Environ Health. 2008 May 16;7:17. doi: 10.1186/1476-069X-7-17.
There is a growing body of literature linking GIS-based measures of traffic density to asthma and other respiratory outcomes. However, no consensus exists on which traffic indicators best capture variability in different pollutants or within different settings. As part of a study on childhood asthma etiology, we examined variability in outdoor concentrations of multiple traffic-related air pollutants within urban communities, using a range of GIS-based predictors and land use regression techniques.
We measured fine particulate matter (PM2.5), nitrogen dioxide (NO2), and elemental carbon (EC) outside 44 homes representing a range of traffic densities and neighborhoods across Boston, Massachusetts and nearby communities. Multiple three to four-day average samples were collected at each home during winters and summers from 2003 to 2005. Traffic indicators were derived using Massachusetts Highway Department data and direct traffic counts. Multivariate regression analyses were performed separately for each pollutant, using traffic indicators, land use, meteorology, site characteristics, and central site concentrations.
PM2.5 was strongly associated with the central site monitor (R2 = 0.68). Additional variability was explained by total roadway length within 100 m of the home, smoking or grilling near the monitor, and block-group population density (R2 = 0.76). EC showed greater spatial variability, especially during winter months, and was predicted by roadway length within 200 m of the home. The influence of traffic was greater under low wind speed conditions, and concentrations were lower during summer (R2 = 0.52). NO2 showed significant spatial variability, predicted by population density and roadway length within 50 m of the home, modified by site characteristics (obstruction), and with higher concentrations during summer (R2 = 0.56).
Each pollutant examined displayed somewhat different spatial patterns within urban neighborhoods, and were differently related to local traffic and meteorology. Our results indicate a need for multi-pollutant exposure modeling to disentangle causal agents in epidemiological studies, and further investigation of site-specific and meteorological modification of the traffic-concentration relationship in urban neighborhoods.
越来越多的文献将基于地理信息系统(GIS)的交通密度测量与哮喘及其他呼吸道疾病结局联系起来。然而,对于哪种交通指标能最好地反映不同污染物或不同环境中的变异性,目前尚无共识。作为一项关于儿童哮喘病因的研究的一部分,我们使用一系列基于GIS的预测因子和土地利用回归技术,研究了城市社区内多种与交通相关的空气污染物的室外浓度变异性。
我们在马萨诸塞州波士顿及附近社区的44户家庭外测量了细颗粒物(PM2.5)、二氧化氮(NO2)和元素碳(EC),这些家庭代表了不同的交通密度和社区类型。在2003年至2005年的冬季和夏季,在每户家庭采集了多个三到四天的平均样本。交通指标通过马萨诸塞州公路部的数据和直接交通流量计数得出。对每种污染物分别进行多变量回归分析,使用交通指标、土地利用、气象、场地特征和中心场地浓度。
PM2.5与中心场地监测值密切相关(R2 = 0.68)。家庭100米范围内的道路总长度、监测器附近的吸烟或烧烤以及街区组人口密度解释了额外的变异性(R2 = 0.76)。EC表现出更大的空间变异性,尤其是在冬季月份,可通过家庭200米范围内的道路长度进行预测。在低风速条件下交通的影响更大,夏季浓度较低(R2 = 0.52)。NO2表现出显著的空间变异性,可通过家庭50米范围内的人口密度和道路长度进行预测,并受场地特征(障碍物)影响,夏季浓度较高(R2 = 0.56)结论:所研究的每种污染物在城市社区内呈现出略有不同的空间模式,并且与当地交通和气象的关系也不同。我们的结果表明,在流行病学研究中需要进行多污染物暴露建模以理清因果因素,并进一步研究城市社区中交通 - 浓度关系的特定场地和气象修正。