Blanc F, Bensman A, Baudon J J
Centre de Pediatrie E. Lesne, Hopital Trousseau, Paris, France.
Pediatr Nephrol. 1991 May;5(3):304-6. doi: 10.1007/BF00867486.
We report a case of severe hypertension in the newborn period due to obstruction of the right renal artery. The baby presented with polyuria leading to dehydration and was found to have hyponatraemia and severe renal salt loss. When sudden malignant hypertension is induced in experimental conditions, a high pressure diuresis and increased angiotensin II production are found. These findings could explain the renal salt loss, notwithstanding the effects of secondary hyperaldosteronism and hyper-reninaemia.
我们报告一例因右肾动脉梗阻导致新生儿期严重高血压的病例。该婴儿出现多尿导致脱水,检查发现有低钠血症和严重肾盐丢失。在实验条件下诱发突然的恶性高血压时,会出现高压利尿和血管紧张素II生成增加。尽管存在继发性醛固酮增多症和高肾素血症的影响,但这些发现可以解释肾盐丢失的原因。