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Distribution of sensitivity to 4-nitroquinoline 1-oxide among Japanese lymphoblastoid cell lines.

作者信息

Kiyohara C, Hirohata T, Kuratsune M, Nagayama J

机构信息

Department of Public Health, Faculty of Medicine, Kyushu University, Fukuoka, Japan.

出版信息

Bull Environ Contam Toxicol. 1991 Jan;46(1):62-8. doi: 10.1007/BF01688255.

DOI:10.1007/BF01688255
PMID:1900452
Abstract
摘要

相似文献

1
Distribution of sensitivity to 4-nitroquinoline 1-oxide among Japanese lymphoblastoid cell lines.日本淋巴母细胞系对4-硝基喹啉-1-氧化物的敏感性分布
Bull Environ Contam Toxicol. 1991 Jan;46(1):62-8. doi: 10.1007/BF01688255.
2
An improved method for the detection of differential survival between normal and xeroderma pigmentosum lymphoblastoid cell lines in culture with 4-nitroquinoline-1-oxide.一种利用4-硝基喹啉-1-氧化物检测培养的正常和着色性干皮病淋巴母细胞系之间差异存活的改进方法。
Mutat Res. 1991 Jul;249(1):111-7. doi: 10.1016/0027-5107(91)90137-d.
3
Spontaneous and 4-nitroquinoline 1-oxide-induced G2 chromosome aberrations in lymphoblasts from familial melanoma patients.家族性黑色素瘤患者淋巴细胞中自发的以及4-硝基喹啉1-氧化物诱导的G2期染色体畸变
Cancer Genet Cytogenet. 1989 Jun;39(2):233-43. doi: 10.1016/0165-4608(89)90190-8.
4
Alkaline sucrose sedimentation analysis as an indicator of repair capability of xeroderma pigmentosum fibroblasts for 4-nitroquinoline-1-oxide damage.碱性蔗糖沉降分析作为着色性干皮病成纤维细胞对4-硝基喹啉-1-氧化物损伤修复能力的指标。
Carcinogenesis. 1981;2(8):691-5. doi: 10.1093/carcin/2.8.691.
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[Mechanisms of impairment of DNA repair in human cells. Interferons stimulated DNA repair in xeroderma pigmentosum cells].[人类细胞中DNA修复受损的机制。干扰素刺激着色性干皮病细胞中的DNA修复]
Genetika. 1989 Sep;25(9):1658-63.
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DNA repair characteristics and skin cancers of xeroderma pigmentosum patients in Japan.日本着色性干皮病患者的DNA修复特征与皮肤癌
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High sensitivity but normal DNA-repair activity after UV irradiation in Epstein--Barr virus-transformed lymphoblastoid cell lines from Chediak--Higashi syndrome.来自切-东综合征患者的爱泼斯坦-巴尔病毒转化淋巴母细胞系在紫外线照射后具有高敏感性但DNA修复活性正常。
Mutat Res. 1980 Aug;72(1):143-50. doi: 10.1016/0027-5107(80)90230-4.
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Assignment of two Japanese xeroderma pigmentosum patients to complementation group D and their characteristics.两名日本着色性干皮病患者被归入互补组D及其特征
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Xeroderma pigmentosum group E binding factor recognizes a broad spectrum of DNA damage.
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Excision of 4-nitroquinoline 1-oxide damage and transformed in mouse cells.
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本文引用的文献

1
Evidence for excision of ultraviolet-induced pyrimidine dimers from the DNA of human cells in vitro.体外人细胞DNA中紫外线诱导嘧啶二聚体切除的证据。
Biophys J. 1968 Mar;8(3):319-25. doi: 10.1016/S0006-3495(68)86490-2.
2
Defective repair replication of DNA in xeroderma pigmentosum.着色性干皮病中DNA修复复制缺陷。
Nature. 1968 May 18;218(5142):652-6. doi: 10.1038/218652a0.
3
Absence of excision of ultraviolet-induced cyclobutane dimers in xeroderma pigmentosum.着色性干皮病中紫外线诱导的环丁烷二聚体切除缺失。
Photochem Photobiol. 1970 Jun;11(6):547-50. doi: 10.1111/j.1751-1097.1970.tb06025.x.
4
Evidence that xeroderma pigmentosum cells do not perform the first step in the repair of ultraviolet damage to their DNA.着色性干皮病细胞在修复其DNA紫外线损伤时不执行第一步的证据。
Proc Natl Acad Sci U S A. 1969 Nov;64(3):1035-41. doi: 10.1073/pnas.64.3.1035.
5
Membrane properties of living mammalian cells as studied by enzymatic hydrolysis of fluorogenic esters.通过荧光酯的酶促水解研究活的哺乳动物细胞的膜特性。
Proc Natl Acad Sci U S A. 1966 Jan;55(1):134-41. doi: 10.1073/pnas.55.1.134.
6
The photochemistry, photobiology, and repair of polynucleotides.多核苷酸的光化学、光生物学及修复
Prog Nucleic Acid Res Mol Biol. 1968;8:257-95. doi: 10.1016/s0079-6603(08)60548-6.
7
Xeroderma pigmentosum. An inherited diseases with sun sensitivity, multiple cutaneous neoplasms, and abnormal DNA repair.着色性干皮病。一种遗传性疾病，对阳光敏感，有多种皮肤肿瘤，且DNA修复异常。
Ann Intern Med. 1974 Feb;80(2):221-48. doi: 10.7326/0003-4819-80-2-221.
8
Interaction of a carcinogen, 4-nitroquinoline-1-oxide, with nucleic acids: chemical degradation of the adducts.致癌物4-硝基喹啉-1-氧化物与核酸的相互作用：加合物的化学降解
Chem Biol Interact. 1971 Jun;3(3):225-9. doi: 10.1016/0009-2797(71)90086-x.
9
A quantitative analysis of the susceptibility of human leukocytes to transformation by Epstein-Barr virus.对人类白细胞受爱泼斯坦-巴尔病毒转化的易感性的定量分析。
Int J Cancer. 1976 Jul 15;18(1):7-13. doi: 10.1002/ijc.2910180103.
10
Seryl-tRNA synthetase and activation of the carcinogen 4-nitroguinoline 1-oxide.
Nature. 1975 Jun 5;255(5508):510-2. doi: 10.1038/255510a0.