Ataka Hiromi, Tanno Takaaki, Yamazaki Masashi
Department of Orthopaedic Surgery, Matsudo City Hospital, Matsudo, Chiba, Japan.
Eur Spine J. 2009 Jan;18(1):69-76. doi: 10.1007/s00586-008-0821-8. Epub 2008 Nov 13.
Previous reports have emphasized the importance of neural decompression through either an anterior or posterior approach when reconstruction surgery is performed for neurological deficits following vertebral collapse in the osteoporotic thoracolumbar spine. However, the contribution of these decompression procedures to neurological recovery has not been fully established. In the present study, we investigated 14 consecutive patients who had incomplete neurological deficits following vertebral collapse in the osteoporotic thoracolumbar spine and underwent posterior instrumented fusion without neural decompression. They were radiographically and neurologically assessed during an average follow-up period of 25 months. The mean local kyphosis angle was 14.6 degrees at flexion and 4.1 degrees at extension preoperatively, indicating marked instability at the collapsed vertebrae. The mean spinal canal occupation by bone fragments was 21%. After surgery, solid bony fusion was obtained in all patients. The mean local kyphosis angle became 5.8 degrees immediately after surgery and 9.9 degrees at the final follow-up. There was no implant dislodgement, and no additional surgery was required. In all patients, back pain was relieved, and neurological improvement was obtained by at least one modified Frankel grade. The present series demonstrate that the posterior instrumented fusion without neural decompression for incomplete neurological deficits following vertebral collapse in the osteoporotic thoracolumbar spine can provide neurological improvement and relief of back pain without major complications. We suggest that neural decompression is not essential for the treatment of neurological impairment due to osteoporotic vertebral collapse with dynamic mobility.
既往报道强调,在对骨质疏松性胸腰椎椎体塌陷后出现神经功能缺损进行重建手术时,通过前路或后路进行神经减压的重要性。然而,这些减压手术对神经功能恢复的作用尚未完全明确。在本研究中,我们调查了14例骨质疏松性胸腰椎椎体塌陷后出现不完全神经功能缺损且接受了后路器械融合但未进行神经减压的连续患者。在平均25个月的随访期内对他们进行了影像学和神经功能评估。术前,平均局部后凸角在屈曲时为14.6度,伸展时为4.1度,表明塌陷椎体存在明显不稳定。骨碎片平均占据椎管的比例为21%。术后,所有患者均获得了坚固的骨融合。术后即刻平均局部后凸角变为5.8度,末次随访时为9.9度。没有植入物移位,也无需再次手术。所有患者的背痛均得到缓解,神经功能至少改善了一个改良Frankel分级。本系列研究表明,对于骨质疏松性胸腰椎椎体塌陷后不完全神经功能缺损,后路器械融合而不进行神经减压可改善神经功能并缓解背痛,且无重大并发症。我们认为,对于伴有动态活动的骨质疏松性椎体塌陷所致神经功能损害的治疗,神经减压并非必不可少。