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胃食管反流病导致的牙侵蚀:一例报告

Dental erosion caused by gastroesophageal reflux disease: a case report.

作者信息

Cengiz Seda, Cengiz M Inanç, Saraç Y Sinasi

机构信息

Prosthetic Dentistry, Private Practice, Samsun, Turkey.

出版信息

Cases J. 2009 Jul 22;2:8018. doi: 10.4076/1757-1626-2-8018.

DOI:10.4076/1757-1626-2-8018
PMID:19830044
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2740145/
Abstract

INTRODUCTION

Chronic regurgitation of gastric acids in patients with gastroesophageal reflux disease may cause dental erosion, which can lead in combination with attrition or bruxism to extensive loss of coronal tooth tissue.

CASE PRESENTATION

This clinical report describes treatment of severe tooth wear of a gastroesophageal reflux disease patient who is 54-year-old Turkish male patient. After his medical treatment, severe tooth wear, bruxism and decreased vertical dimensions were determined. The vertical dimension was re-established and maxillary and mandibular anterior and posterior teeth were prepared for metal-ceramic restorations. Metal-ceramic fixed partial dentures were fabricated as full mouth restorations for both maxillary and mandibular arches because of splinting all teeth. And then maxillary stabilization splint was fabricated for his bruxism history.

CONCLUSION

Significant loss of coronal tooth structure must taken into consideration. Gastroesophageal reflux disease by itself or in combination with attrition, abrasion or bruxism may be responsible for the loss. An extensive diagnostic evaluation is essential for the medical and dental effects of the problem.

摘要

引言

胃食管反流病患者胃酸的慢性反流可能导致牙齿侵蚀,这与磨耗或磨牙症相结合可导致牙冠牙齿组织的广泛丧失。

病例报告

本临床报告描述了一名54岁土耳其男性胃食管反流病患者严重牙齿磨损的治疗情况。在他接受药物治疗后,发现有严重的牙齿磨损、磨牙症和垂直距离减小。重新建立了垂直距离,并对上颌和下颌的前后牙进行了金属烤瓷修复体的预备。由于需要固定所有牙齿,因此制作了金属烤瓷固定局部义齿作为上颌和下颌牙弓的全口修复体。然后,鉴于他有磨牙症病史,制作了上颌稳定夹板。

结论

必须考虑到牙冠牙齿结构的显著丧失。胃食管反流病本身或与磨耗、磨损或磨牙症相结合可能是导致这种丧失的原因。对于该问题的医学和牙科影响,进行广泛的诊断评估至关重要。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/a12ea9f417b2/1757-1626-0002-0000008018-009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/d46db21ac621/1757-1626-0002-0000008018-001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/2e663f62127e/1757-1626-0002-0000008018-002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/5f9367bb503c/1757-1626-0002-0000008018-003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/f8aaac40bfc1/1757-1626-0002-0000008018-004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/3e6eb2de6091/1757-1626-0002-0000008018-005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/f9ee994187d7/1757-1626-0002-0000008018-006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/48a26a357a61/1757-1626-0002-0000008018-007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/eee42a25b6ec/1757-1626-0002-0000008018-008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/a12ea9f417b2/1757-1626-0002-0000008018-009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/d46db21ac621/1757-1626-0002-0000008018-001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/2e663f62127e/1757-1626-0002-0000008018-002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/5f9367bb503c/1757-1626-0002-0000008018-003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/f8aaac40bfc1/1757-1626-0002-0000008018-004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/3e6eb2de6091/1757-1626-0002-0000008018-005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/f9ee994187d7/1757-1626-0002-0000008018-006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/48a26a357a61/1757-1626-0002-0000008018-007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/eee42a25b6ec/1757-1626-0002-0000008018-008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f13a/2740145/a12ea9f417b2/1757-1626-0002-0000008018-009.jpg

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