抗痛素（一种蛋白酶抑制剂）对紫外线照射的大肠杆菌细胞呼吸、活力及嘧啶二聚体切除的影响。
Effects of antipain (a protease inhibitor) on respiration, viability, and excision of pyrimidine dimers in UV-irradiated Escherichia coli cells.
作者信息
Swenson P A, Schenley R L
出版信息
J Bacteriol. 1978 Sep;135(3):1167-70. doi: 10.1128/jb.135.3.1167-1170.1978.
Abstract
The protease inhibitor antipain increases the effectiveness of UV irradiation on cessation of respiration and cell killing in Escherichia coli B/r cultures without affecting excision of pyrimidine dimers. The actions are similar to those caused by cyclic AMP in irradiated cultures.
摘要
蛋白酶抑制剂抗痛素可提高紫外线照射对大肠杆菌B/r培养物呼吸停止和细胞杀伤的效果，且不影响嘧啶二聚体的切除。这些作用与环磷酸腺苷在受照射培养物中所引起的作用相似。
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本文引用的文献
1
Structure of antipain, a new Sakaguchi-positive product of streptomyces.抗痛素的结构，链霉菌一种新的坂口反应阳性产物
J Antibiot (Tokyo). 1972 Apr;25(4):267-70. doi: 10.7164/antibiotics.25.267.
2
Evidence relating cessation of respiration, cell envelope changes, and death in ultraviolet-irradiated Escherichia coli B-r cells.关于紫外线照射的大肠杆菌B-r细胞中呼吸停止、细胞包膜变化和死亡的相关证据。
J Bacteriol. 1974 Feb;117(2):551-9. doi: 10.1128/jb.117.2.551-559.1974.
3
Respiration, growth and viability of repair-deficient mutants of Escherichia coli after ultraviolet irradiation.紫外线照射后大肠杆菌修复缺陷型突变体的呼吸作用、生长及生存能力
Int J Radiat Biol Relat Stud Phys Chem Med. 1974 Jan;25(1):51-60. doi: 10.1080/09553007414550051.
4
Ultraviolet mutagenesis and inducible DNA repair in Escherichia coli.大肠杆菌中的紫外线诱变与诱导性DNA修复
Bacteriol Rev. 1976 Dec;40(4):869-907. doi: 10.1128/br.40.4.869-907.1976.
5
Centrifugal separation of irradiated cultures of Escherichia coli cells into viable and nonviable populations.将经辐照的大肠杆菌细胞培养物离心分离成活细胞群体和非活细胞群体。
J Bacteriol. 1976 May;126(2):977-84. doi: 10.1128/jb.126.2.977-984.1976.
6
Inactivation and proteolytic cleavage of phage lambda repressor in vitro in an ATP-dependent reaction.在依赖ATP的反应中，噬菌体λ阻遏物在体外的失活和蛋白水解切割。
Proc Natl Acad Sci U S A. 1977 Jun;74(6):2283-7. doi: 10.1073/pnas.74.6.2283.
7
A protease inhibitor blocks SOS functions in Escherichia coli: antipain prevents lambda repressor inactivation, ultraviolet mutagenesis, and filamentous growth.一种蛋白酶抑制剂可阻断大肠杆菌中的SOS功能：抗蛋白酶可防止λ阻遏物失活、紫外线诱变及丝状生长。
Proc Natl Acad Sci U S A. 1977 Mar;74(3):1152-6. doi: 10.1073/pnas.74.3.1152.
8
Regulation of cessation of respiration and killing by cyclic 3',5'-adenosine monophosphate and its receptor protein after far-ultraviolet irradiation of Escherichia coli.
Mol Gen Genet. 1978 Feb 16;159(2):125-30. doi: 10.1007/BF00270885.
9
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J Bacteriol. 1977 Aug;131(2):707-9. doi: 10.1128/jb.131.2.707-709.1977.
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