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1
Persistent nephrogenic diabetes insipidus, tubular proteinuria, aminoaciduria, and parathyroid hormone resistance following longterm lithium administration.长期服用锂剂后出现持续性肾性尿崩症、肾小管性蛋白尿、氨基酸尿和甲状旁腺激素抵抗。
Postgrad Med J. 1990 Jun;66(776):479-82. doi: 10.1136/pgmj.66.776.479.
2
Lithium and the kidney.锂与肾脏
N Z Med J. 1979 Oct 24;90(646):323-5.
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Lithium-induced nephrogenic diabetes insipidus: studies of tubular function and pathogenesis.锂诱导的肾性尿崩症：肾小管功能及发病机制研究
Isr J Med Sci. 1979 Sep;15(9):765-71.
4
Lithium nephrotoxicity: a review.锂的肾毒性：综述
Ann Clin Lab Sci. 1981 Jul-Aug;11(4):343-9.
5
Lithium and the kidney: a review.锂与肾脏：综述
Am J Psychiatry. 1982 Apr;139(4):443-9. doi: 10.1176/ajp.139.4.443.
6
Prevalence, pathogenesis, and treatment of renal dysfunction associated with chronic lithium therapy.与慢性锂盐治疗相关的肾功能不全的患病率、发病机制及治疗
Am J Kidney Dis. 1987 Nov;10(5):329-45. doi: 10.1016/s0272-6386(87)80098-7.
7
[Impaired kidney function in lithium therapy].[锂治疗中的肾功能损害]
Ther Umsch. 1998 Sep;55(9):562-4.
8
Lithium carbonate-induced nephrogenic diabetes insipidus and glucose intolerance.碳酸锂所致的肾性尿崩症和葡萄糖不耐受。
Arch Intern Med. 1973 Dec;132(6):881-4.
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[Nephrotoxicity and long-term treatment with lithium].
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Persistent nephrogenic diabetes insipidus following lithium therapy.锂治疗后持续性肾性尿崩症
Scott Med J. 1997 Feb;42(1):16-7. doi: 10.1177/003693309704200106.

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Prolonged hypernatremia triggered by hyperglycemic hyperosmolar state with coma: A case report.高血糖高渗状态伴昏迷引发的持续性高钠血症：一例报告
World J Nephrol. 2015 May 6;4(2):319-23. doi: 10.5527/wjn.v4.i2.319.

本文引用的文献

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THE USE OF LITHIUM SALTS IN AFFECTIVE DISORDERS.锂盐在情感障碍中的应用。
Br J Psychiatry. 1963 Nov;109:810-4. doi: 10.1192/bjp.109.463.810.
2
Lithium treatment and kidney function. A follow-up study of 237 patients in long-term treatment.锂治疗与肾功能。对237例长期治疗患者的随访研究。
Acta Psychiatr Scand. 1981 Apr;63(4):333-45. doi: 10.1111/j.1600-0447.1981.tb00682.x.
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The relationship between plasma lithium and the renal responsiveness to arginine vasopressin in man.人体血浆锂与肾脏对精氨酸加压素反应性之间的关系。
Clin Sci (Lond). 1981 Dec;61(6):793-5. doi: 10.1042/cs0610793.
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Lithium and the kidney: a review.锂与肾脏：综述
Am J Psychiatry. 1982 Apr;139(4):443-9. doi: 10.1176/ajp.139.4.443.
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Impairment of renal function in patients on long-term lithium treatment.长期接受锂盐治疗患者的肾功能损害
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6
Lithium induced interstitial nephropathy associated with chronic renal failure. Reversibility and correlation between functional and structural changes.锂诱导的间质性肾病伴慢性肾衰竭。功能与结构改变之间的可逆性及相关性。
Acta Pathol Microbiol Immunol Scand A. 1984 Nov;92(6):447-54. doi: 10.1111/j.1699-0463.1984.tb04426.x.
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Characterization of acidification in the cortical and medullary collecting tubule of the rabbit.兔皮质和髓质集合管酸化作用的特征
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8
Distal nephron function in patients receiving chronic lithium therapy.接受慢性锂盐治疗患者的远端肾单位功能
Kidney Int. 1982 Mar;21(3):477-85. doi: 10.1038/ki.1982.49.
9
Vasopressin-resistant hyposthenuria in advanced chronic renal disease.晚期慢性肾病中的抗血管加压素性低渗尿
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Metabolic balance studies on the effect of lithium salts in manic-depressive psychosis.
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长期服用锂剂后出现持续性肾性尿崩症、肾小管性蛋白尿、氨基酸尿和甲状旁腺激素抵抗。

Persistent nephrogenic diabetes insipidus, tubular proteinuria, aminoaciduria, and parathyroid hormone resistance following longterm lithium administration.

作者信息

Neithercut W D, Spooner R J, Hendry A, Dagg J H

机构信息

Department of Pathological Biochemistry, Western Infirmary, Glasgow, UK.

出版信息

Postgrad Med J. 1990 Jun;66(776):479-82. doi: 10.1136/pgmj.66.776.479.

DOI:10.1136/pgmj.66.776.479
PMID:2170960
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2429604/
Abstract

We report a patient who developed persistent nephrogenic diabetes insipidus associated with renal tubular acidosis, renal resistance to parathyroid hormone, aminoaciduria and proximal tubule pattern proteinuria in the presence of a reduced glomerular filtration rate (19-24 ml/min). A review of the previous reports of persistent nephrogenic diabetes insipidus revealed that in all patients the glomerular filtration rate had been less than 60 ml/min at presentation. Chronic renal failure may therefore predispose to the development of persistent nephrogenic diabetes insipidus in patients receiving lithium.

摘要

我们报告了一名患者，该患者在肾小球滤过率降低（19 - 24 ml/分钟）的情况下，出现了与肾小管酸中毒、肾对甲状旁腺激素抵抗、氨基酸尿和近端肾小管型蛋白尿相关的持续性肾性尿崩症。对既往持续性肾性尿崩症报告的回顾显示，所有患者在就诊时肾小球滤过率均低于60 ml/分钟。因此，慢性肾衰竭可能使接受锂治疗的患者易患持续性肾性尿崩症。