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Macrophages, atherosclerosis and the potential of netrin-1 as a novel target for future therapeutic intervention.巨噬细胞、动脉粥样硬化以及netrin-1作为未来治疗干预新靶点的潜力。
Future Cardiol. 2012 May;8(3):349-52. doi: 10.2217/fca.12.30.
2
The neuroimmune guidance cue netrin-1 promotes atherosclerosis by inhibiting the emigration of macrophages from plaques.神经免疫导向分子 netrin-1 通过抑制斑块内巨噬细胞的迁移来促进动脉粥样硬化的形成。
Nat Immunol. 2012 Jan 8;13(2):136-43. doi: 10.1038/ni.2205.
3
TNF-α mediates macrophage-induced bystander effects through Netrin-1.TNF-α 通过 Netrin-1 介导巨噬细胞诱导的旁观者效应。
Cancer Res. 2012 Oct 15;72(20):5219-29. doi: 10.1158/0008-5472.CAN-12-1463. Epub 2012 Aug 22.
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Inhibition of DNA methylation promotes breast tumor sensitivity to netrin-1 interference.DNA甲基化的抑制促进乳腺肿瘤对netrin-1干扰的敏感性。
EMBO Mol Med. 2016 Aug 1;8(8):863-77. doi: 10.15252/emmm.201505945. Print 2016 Aug.
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Netrin-1: A regulator of cancer cell motility?轴突导向因子 1：癌细胞运动的调节因子？
Eur J Cell Biol. 2016 Nov;95(11):513-520. doi: 10.1016/j.ejcb.2016.10.002. Epub 2016 Oct 19.
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Plasma netrin-1 is a diagnostic biomarker of human cancers.血浆 netrin-1 是人类癌症的诊断生物标志物。
Biomarkers. 2011 Mar;16(2):172-80. doi: 10.3109/1354750X.2010.541564. Epub 2011 Feb 8.
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Netrin-1 is associated with macrophage infiltration and polarization in human epicardial adipose tissue in coronary artery disease.Netrin-1与冠状动脉疾病患者心外膜脂肪组织中的巨噬细胞浸润和极化有关。
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AAV-mediated netrin-1 overexpression increases peri-infarct blood vessel density and improves motor function recovery after experimental stroke.腺相关病毒介导的轴突导向因子 1 过表达增加实验性卒中后梗死周围血管密度并改善运动功能恢复。
Neurobiol Dis. 2011 Oct;44(1):73-83. doi: 10.1016/j.nbd.2011.06.006. Epub 2011 Jun 25.
9
Expression of Netrin-1 and its two receptors DCC and UNC5H2 in the developing mouse lung.Netrin-1及其两种受体DCC和UNC5H2在发育中的小鼠肺中的表达。
Gene Expr Patterns. 2003 Jun;3(3):279-83. doi: 10.1016/s1567-133x(03)00047-4.
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Cytoprotective and proangiogenic activity of ex-vivo netrin-1 transgene overexpression protects the heart against ischemia/reperfusion injury.外源性 netrin-1 过表达的细胞保护和促血管生成活性可保护心脏免受缺血/再灌注损伤。
Stem Cells Dev. 2012 Jul 1;21(10):1769-78. doi: 10.1089/scd.2011.0475. Epub 2011 Nov 11.

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Long-Term Prognostic Value of Adipocytokines in Patients with Acute Coronary Syndrome: An 8-Year Clinical Prospective Cohort Study.脂肪细胞因子对急性冠脉综合征患者的长期预后价值：一项为期8年的临床前瞻性队列研究
J Inflamm Res. 2024 Oct 2;17:6989-7003. doi: 10.2147/JIR.S483600. eCollection 2024.
2
Netrin-1 and RGMa: Novel Regulators of Atherosclerosis-Related Diseases.Netrin-1与RGMa：动脉粥样硬化相关疾病的新型调节因子
Cardiovasc Drugs Ther. 2025 Feb;39(1):211-219. doi: 10.1007/s10557-023-07478-5. Epub 2023 Jul 13.
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Netrin-1 and its receptor Unc5b are novel targets for the treatment of inflammatory arthritis.Netrin-1及其受体Unc5b是治疗炎性关节炎的新靶点。
FASEB J. 2016 Nov;30(11):3835-3844. doi: 10.1096/fj.201600615R. Epub 2016 Aug 8.
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Evaluation of Netrin-1 Levels and Albuminuria in Patients With Diabetes.糖尿病患者中Netrin-1水平与蛋白尿的评估
J Clin Lab Anal. 2016 Nov;30(6):972-977. doi: 10.1002/jcla.21965. Epub 2016 Apr 13.
5
The effect of prebiotic supplementation with inulin on cardiometabolic health: Rationale, design, and methods of a controlled feeding efficacy trial in adults at risk of type 2 diabetes.补充菊粉型益生元对心脏代谢健康的影响：2型糖尿病风险成年人群对照喂养疗效试验的理论依据、设计与方法
Contemp Clin Trials. 2015 Nov;45(Pt B):328-337. doi: 10.1016/j.cct.2015.10.012. Epub 2015 Oct 28.
6
Molecular sources of residual cardiovascular risk, clinical signals, and innovative solutions: relationship with subclinical disease, undertreatment, and poor adherence: implications of new evidence upon optimizing cardiovascular patient outcomes.残余心血管风险的分子来源、临床信号及创新解决方案：与亚临床疾病、治疗不足及依从性差的关系：新证据对优化心血管疾病患者预后的影响
Vasc Health Risk Manag. 2013;9:617-70. doi: 10.2147/VHRM.S37119. Epub 2013 Oct 21.

本文引用的文献

1
The neuroimmune guidance cue netrin-1 promotes atherosclerosis by inhibiting the emigration of macrophages from plaques.神经免疫导向分子 netrin-1 通过抑制斑块内巨噬细胞的迁移来促进动脉粥样硬化的形成。
Nat Immunol. 2012 Jan 8;13(2):136-43. doi: 10.1038/ni.2205.
2
Inhibition of miR-33a/b in non-human primates raises plasma HDL and lowers VLDL triglycerides.在非人类灵长类动物中抑制 miR-33a/b 可提高血浆高密度脂蛋白胆固醇并降低极低密度脂蛋白甘油三酯。
Nature. 2011 Oct 19;478(7369):404-7. doi: 10.1038/nature10486.
3
Antagonism of miR-33 in mice promotes reverse cholesterol transport and regression of atherosclerosis.在小鼠体内拮抗 miR-33 可促进胆固醇逆向转运并使动脉粥样硬化消退。
J Clin Invest. 2011 Jul;121(7):2921-31. doi: 10.1172/JCI57275. Epub 2011 Jun 6.
4
Macrophages in the pathogenesis of atherosclerosis.动脉粥样硬化发病机制中的巨噬细胞。
Cell. 2011 Apr 29;145(3):341-55. doi: 10.1016/j.cell.2011.04.005.
5
Suppressed monocyte recruitment drives macrophage removal from atherosclerotic plaques of Apoe-/- mice during disease regression.在动脉粥样硬化斑块消退期间，受抑制的单核细胞募集导致 Apoe-/- 小鼠的巨噬细胞从斑块中被清除。
J Clin Invest. 2011 May;121(5):2025-36. doi: 10.1172/JCI43802. Epub 2011 Apr 18.
6
Reversal of hyperlipidemia with a genetic switch favorably affects the content and inflammatory state of macrophages in atherosclerotic plaques.通过基因开关逆转高血脂可改善动脉粥样硬化斑块中巨噬细胞的含量和炎症状态。
Circulation. 2011 Mar 8;123(9):989-98. doi: 10.1161/CIRCULATIONAHA.110.984146. Epub 2011 Feb 21.
7
NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals.NLRP3 炎性小体对于动脉粥样硬化的形成是必需的，并且可以被胆固醇晶体激活。
Nature. 2010 Apr 29;464(7293):1357-61. doi: 10.1038/nature08938.
8
Macrophage death and defective inflammation resolution in atherosclerosis.动脉粥样硬化中的巨噬细胞死亡和炎症消退缺陷。
Nat Rev Immunol. 2010 Jan;10(1):36-46. doi: 10.1038/nri2675. Epub 2009 Dec 4.
9
Hypoxia-inducible factor-dependent induction of netrin-1 dampens inflammation caused by hypoxia.缺氧诱导因子依赖性的网蛋白-1诱导可减轻缺氧引起的炎症。
Nat Immunol. 2009 Feb;10(2):195-202. doi: 10.1038/ni.1683. Epub 2009 Jan 4.
10
CD36 modulates migration of mouse and human macrophages in response to oxidized LDL and may contribute to macrophage trapping in the arterial intima.CD36可调节小鼠和人类巨噬细胞对氧化型低密度脂蛋白的迁移反应，并可能促使巨噬细胞滞留于动脉内膜。
J Clin Invest. 2009 Jan;119(1):136-45. doi: 10.1172/JCI35535. Epub 2008 Dec 8.

Macrophages, atherosclerosis and the potential of netrin-1 as a novel target for future therapeutic intervention.

作者信息

Moore Kathryn J, Fisher Edward A

出版信息

Future Cardiol. 2012 May;8(3):349-52. doi: 10.2217/fca.12.30.

DOI:10.2217/fca.12.30
PMID:22642628
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3920546/
Abstract
摘要