炎症性关节积液中淋巴细胞内单纯疱疹病毒的命运。ii. 非允许性机制。
Fate of herpes simplex virus in lymphocytes from inflammatory joint effusions. ii. Mechanisms of non-permissiveness.
作者信息
Appleford D J, Denman A M
出版信息
Ann Rheum Dis. 1979 Oct;38(5):450-5. doi: 10.1136/ard.38.5.450.
Abstract
Mononuclear cells which were isolated from rheumatoid synovial effusions and cultured without prior separation failed to support the growth of herpes simplex virus (HSV). However, subpopulations of T lymphocytes separated on the basis of cell size or receptors for sheep red cells were, in isolation, permissive for this virus. Virus growth in these permissive cells was blocked by cocultivation with the remaining mononuclear cell populations.
摘要
从类风湿性滑膜炎积液中分离出的单核细胞，未经预先分离就进行培养，无法支持单纯疱疹病毒（HSV）的生长。然而，根据细胞大小或绵羊红细胞受体分离出的T淋巴细胞亚群，单独培养时对该病毒具有易感性。在这些易感细胞中的病毒生长，会因与其余单核细胞群体共同培养而受到抑制。
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本文引用的文献
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Separation of cells by velocity sedimentation.通过速度沉降法分离细胞。
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Lymphocyte activation. I. Response of T and B lymphocytes to phytomitogens.淋巴细胞活化。I. T淋巴细胞和B淋巴细胞对植物有丝分裂原的反应。
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Enhanced binding of neuraminidase-treated sheep erythrocytes to human T lymphocytes.神经氨酸酶处理的绵羊红细胞与人类T淋巴细胞的结合增强。
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Characterization of mononuclear effector cells in human blood.人血中单核效应细胞的特性分析
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Antiviral protection by virus-immune cytotoxic T cells: infected target cells are lysed before infectious virus progeny is assembled.病毒免疫细胞毒性T细胞提供的抗病毒保护：在感染性病毒后代组装之前，被感染的靶细胞就会被裂解。
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