痛风性关节炎急性炎症机制的当前概念。
Current concepts of the mechanism of acute inflammation in gouty arthritis.
作者信息
Spilberg I
出版信息
Arthritis Rheum. 1975 Mar-Apr;18(2):129-34. doi: 10.1002/art.1780180208.
PMID:236759
Abstract
The acute gouty attack develops after free crystalline monosodium urate crystals appear in the joint cavity (1). Recent developments in the investigation of urate crystal-induced inflammation have led to a better understanding of the pathogenesis of acute gouty arthritis. The purpose of this review is to summarize present concepts concerning the mechanism of the acute gouty attack.
摘要
在关节腔中出现游离的尿酸单钠晶体后，会发生急性痛风发作（1）。尿酸盐晶体诱导炎症的研究进展使人们对急性痛风性关节炎的发病机制有了更好的理解。本综述的目的是总结目前关于急性痛风发作机制的概念。
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