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与停训相关的年轻运动员左心室壁厚度和纵向应变的变化,可能与肥厚型心肌病有关。

Detraining-related changes in left ventricular wall thickness and longitudinal strain in a young athlete likely to have hypertrophic cardiomyopathy.

机构信息

Clinical and Experimental Department of Medicine and Pharmacology, University Hospital of Messina , Messina, Italy.

出版信息

J Sports Sci Med. 2012 Sep 1;11(3):557-61. eCollection 2012.

Abstract

One of the diagnostic criteria in order to differentiate between physiological and pathological left ventricular hypertrophy is the wall thickness reduction after at least 3-month detraining period, which is considered a marker of the athlete's heart. This report describes detraining-related regression of LV hypertrophy and improvement in myocardial deformation in a junior athlete likely to have hypertrophic cardiomyopathy. Key pointsHypertrophic cardiomyopathy in adolescent athletes can be discovered by 12-lead ECGPhysical training is an important trigger for the clinical presentation of hypertrophic cardiomyopathyReverse LV remodeling (wall thickness reduction) with detraining is a common echocardiographic finding in athletes with physiological hypertrophyThis report demonstrates that reverse remodeling can also be found in adolescent athletes likely to have hypertrophic cardiomyopathy.

摘要

将左心室肥厚分为生理性和病理性的诊断标准之一是,在至少 3 个月的停训期后,壁厚度减少,这被认为是运动员心脏的一个标志。本报告描述了一位年轻运动员在停训后左心室肥厚消退和心肌变形改善,该运动员可能患有肥厚型心肌病。

关键点

  • 青少年运动员中的肥厚型心肌病可通过 12 导联心电图发现

  • 体育训练是肥厚型心肌病临床表现的一个重要诱因

  • 停训后左心室重构(壁厚度减少)是生理性肥厚运动员的常见超声心动图表现

  • 本报告表明,反向重构也可能发生在患有肥厚型心肌病的青少年运动员中。

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