关于过氧化物酶体起源于内质网及其与线粒体关系的进化考量。 - Suppr

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Evolutionary considerations on the origin of peroxisomes from the endoplasmic reticulum, and their relationships with mitochondria.

作者信息

Gabaldón Toni

机构信息

Bioinformatics and Genomics Programme, Centre for Genomic Regulation (CRG), Dr. Aiguader, 88, 08003, Barcelona, Spain,

出版信息

Cell Mol Life Sci. 2014 Jul;71(13):2379-82. doi: 10.1007/s00018-014-1640-1. Epub 2014 May 17.

DOI:10.1007/s00018-014-1640-1

PMID:24838097

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4056328/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/e9de/11113606/0886d8758d1e/18_2014_1640_Fig1_HTML.jpg

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Cell Mol Life Sci. 2014 Jul;71(13):2373-6. doi: 10.1007/s00018-013-1424-z. Epub 2013 Jul 25.

Oxygen radicals shaping evolution: why fatty acid catabolism leads to peroxisomes while neurons do without it: FADH₂/NADH flux ratios determining mitochondrial radical formation were crucial for the eukaryotic invention of peroxisomes and catabolic tissue differentiation.氧自由基塑造进化：脂肪酸分解代谢为何导致过氧化物酶体，而神经元却没有：FADH₂/NADH 通量比决定了线粒体自由基的形成，这对于真核生物过氧化物酶体和分解代谢组织分化的发明至关重要。

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