Skorina I A, Mazing Iu A, Samoĭlova K A, Pigarevskiĭ V E
Tsitologiia. 1988 May;30(5):616-22.
A study was made of the influence of UV-irradiation (254 nm) of blood in vitro, of the autotransfusion of UV-irradiated blood (AUVIB), and of the mixture of UV-irradiated and intact blood in vitro on the content of bactericidal cation proteins (CP) in blood neutrophil of calves suffered from dyspepsia and broncho-pneumonia. Age differences were noticed in CP contents and their decrease in neutrophils following AUVIB in vivo and administration of the mixture of blood in vitro. The decrease in cell CP contents is presumably due to neutrophil degranulation and CP release into the blood plasma. Since the initial mechanisms of neutrophil degranulation are located on the cell surface, the CP release is supposed to result from a membranotropic effect of UV-irradiated blood on the intact autologous blood. This effect may explain the increase in nonspecific resistance of organism after the AUVIB, being one of the main therapeutic phenomena of the AUVIB-therapy.
对体外紫外线（254nm）照射血液、紫外线照射血液自体输血（AUVIB）以及体外紫外线照射血液与未照射血液混合物对患消化不良和支气管肺炎犊牛血液中性粒细胞中杀菌阳离子蛋白（CP）含量的影响进行了研究。观察到CP含量存在年龄差异，以及体内AUVIB和体外血液混合物给药后中性粒细胞中CP含量的降低。细胞CP含量的降低可能是由于中性粒细胞脱颗粒以及CP释放到血浆中。由于中性粒细胞脱颗粒的初始机制位于细胞表面，CP的释放被认为是紫外线照射血液对完整自体血液的膜向性作用所致。这种作用可能解释了AUVIB后机体非特异性抵抗力的增强，这是AUVIB疗法的主要治疗现象之一。
