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Microglia under psychosocial stressors along the aging trajectory: Consequences on neuronal circuits, behavior, and brain diseases.衰老轨迹中心理社会压力下的小胶质细胞：对神经元回路、行为和脑部疾病的影响。
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Infectious immunity in the central nervous system and brain function.中枢神经系统中的感染性免疫与脑功能。
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Postmortem evidence of cerebral inflammation in schizophrenia: a systematic review.精神分裂症脑内炎症的尸检证据：一项系统综述。
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Transgenerational transmission and modification of pathological traits induced by prenatal immune activation.产前免疫激活诱导的病理性特征的跨代传递和修饰。
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Stress and neuroinflammation: a systematic review of the effects of stress on microglia and the implications for mental illness.压力与神经炎症：压力对小胶质细胞影响及其对精神疾病影响的系统综述
Psychopharmacology (Berl). 2016 May;233(9):1637-50. doi: 10.1007/s00213-016-4218-9. Epub 2016 Feb 5.
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Inflammation is associated with decreased functional connectivity within corticostriatal reward circuitry in depression.炎症与抑郁症患者皮质纹状体奖赏回路内功能连接性降低有关。
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PRENATAL INFECTION, MATERNAL IMMUNE ACTIVATION, AND RISK FOR SCHIZOPHRENIA.产前感染、母体免疫激活与精神分裂症风险
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Chronic interferon-α decreases dopamine 2 receptor binding and striatal dopamine release in association with anhedonia-like behavior in nonhuman primates.慢性干扰素-α降低多巴胺 2 受体结合和纹状体多巴胺释放，与非人灵长类动物的快感缺失样行为有关。
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Most genome-wide significant susceptibility loci for schizophrenia and bipolar disorder reported to date cross-traditional diagnostic boundaries.迄今为止报道的大多数精神分裂症和双相情感障碍的全基因组显著易感基因座跨越传统的诊断界限。
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神经炎症：神经精神疾病研究的新视野

Neuroinflammation: new vistas for neuropsychiatric research.

作者信息

Thibaut Florence

机构信息

University Hospital Cochin (Site Tarnier), Faculty of Medicine Paris Descartes, INSERM U 894, CNP, Paris, France.

出版信息

Dialogues Clin Neurosci. 2017 Mar;19(1):3-4. doi: 10.31887/DCNS.2017.19.1/fthibaut.

DOI:10.31887/DCNS.2017.19.1/fthibaut
PMID:28566942
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5442362/
Abstract

In psychiatric diseases such as mood disorders or schizophrenia, the inflammatory response system is activated. Microglia has gradually emerged as a key interface between stress-related signals and neuroimnune consequences of stress, with stressors leading to elevated microglial activity.

摘要

在诸如情绪障碍或精神分裂症等精神疾病中，炎症反应系统被激活。小胶质细胞已逐渐成为应激相关信号与应激的神经免疫后果之间的关键界面，应激源会导致小胶质细胞活性升高。