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Reply: The expanding neurological phenotype of DNM1L-related disorders.回复：与DNM1L相关疾病不断扩展的神经学表型。
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Inhibition of Fatty Acid Metabolism Increases EPA and DHA Levels and Protects against Myocardial Ischaemia-Reperfusion Injury in Zucker Rats.抑制脂肪酸代谢可提高 EPA 和 DHA 水平，并防止 Zucker 大鼠心肌缺血再灌注损伤。
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Effect of Titanium Dioxide and Silver Nanoparticles on Mitochondrial Dynamics in Mouse Testis Tissue.二氧化钛和银纳米粒子对小鼠睾丸组织线粒体动态的影响。
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Metabolic Flexibility of Mitochondria Plays a Key Role in Balancing Glucose and Fatty Acid Metabolism in the Diabetic Heart.线粒体的代谢灵活性在平衡糖尿病心脏中的葡萄糖和脂肪酸代谢方面起着关键作用。
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本文引用的文献

1
GRK2 moderates the acute mitochondrial damage to ionizing radiation exposure by promoting mitochondrial fission/fusion.GRK2通过促进线粒体分裂/融合来减轻电离辐射暴露引起的急性线粒体损伤。
Cell Death Discov. 2018 Feb 14;4:25. doi: 10.1038/s41420-018-0028-7. eCollection 2018 Dec.
2
Enhancing fatty acid utilization ameliorates mitochondrial fragmentation and cardiac dysfunction via rebalancing optic atrophy 1 processing in the failing heart.增强脂肪酸利用可通过重新平衡视蛋白 1 在衰竭心脏中的处理来改善线粒体碎片化和心脏功能障碍。
Cardiovasc Res. 2018 Jun 1;114(7):979-991. doi: 10.1093/cvr/cvy052.
3
New Insights in Cardiac Calcium Handling and Excitation-Contraction Coupling.心脏钙处理和兴奋-收缩偶联的新见解。
Adv Exp Med Biol. 2018;1067:373-385. doi: 10.1007/5584_2017_106.
4
Metabolic Origins of Heart Failure.心力衰竭的代谢起源
JACC Basic Transl Sci. 2017 Jun;2(3):297-310. doi: 10.1016/j.jacbts.2016.11.009.
5
Mechanistic Role of Kinases in the Regulation of Mitochondrial Fitness.激酶在调节线粒体健康中的机制作用
Adv Exp Med Biol. 2017;982:521-528. doi: 10.1007/978-3-319-55330-6_26.
6
Dietary management of heart failure: room for improvement?心力衰竭的饮食管理：是否有改进空间？
Br J Nutr. 2016 Apr 14;115(7):1202-17. doi: 10.1017/S000711451500553X. Epub 2016 Feb 9.
7
Imbalanced OPA1 processing and mitochondrial fragmentation cause heart failure in mice.OPA1 加工失衡和线粒体碎片化导致小鼠心力衰竭。
Science. 2015 Dec 4;350(6265):aad0116. doi: 10.1126/science.aad0116.
8
Meta-analysis of the relation of body mass index to all-cause and cardiovascular mortality and hospitalization in patients with chronic heart failure.慢性心力衰竭患者体重指数与全因死亡率、心血管死亡率及住院率关系的Meta分析
Am J Cardiol. 2015 May 15;115(10):1428-34. doi: 10.1016/j.amjcard.2015.02.024. Epub 2015 Feb 18.
9
Glucose-insulin-potassium revived: current status in acute coronary syndromes and the energy-depleted heart.葡萄糖-胰岛素-钾疗法复苏：急性冠脉综合征及能量耗竭心脏的当前状况
Circulation. 2013 Mar 5;127(9):1040-8. doi: 10.1161/CIRCULATIONAHA.112.130625.
10
Cardiac-specific deletion of acetyl CoA carboxylase 2 prevents metabolic remodeling during pressure-overload hypertrophy.心脏特异性敲除乙酰辅酶 A 羧化酶 2 可防止压力超负荷肥厚期间的代谢重塑。
Circ Res. 2012 Aug 31;111(6):728-38. doi: 10.1161/CIRCRESAHA.112.268128. Epub 2012 Jun 22.

Dietary fat is a key determinant in balancing mitochondrial dynamics in heart failure: a novel mechanism underlying the obesity paradox.

作者信息

De Rosa Matteo, Gambardella Jessica, Shu Jun, Santulli Gaetano

机构信息

Department of Advanced Biomedical Sciences, 'Federico II' University of Naples, Azienda Ospedaliera Universitaria Policlinico, Via Sergio Pansini, 5, Naples 80131, Italy.

Department of Medicine, Albert Einstein College of Medicine, Montefiore University Hospital, 1300 Morris Park Avenue, Forchheimer Medical Science Building, New York, NY 10461, USA.

出版信息

Cardiovasc Res. 2018 Jun 1;114(7):925-927. doi: 10.1093/cvr/cvy074.

DOI:10.1093/cvr/cvy074
PMID:29579150
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5967494/
Abstract
摘要