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[肉毒中毒时脊髓中钠钾ATP酶活性的药物校正可能性]

[Possibilities of pharmacologic correction of Na, K-ATPase activity in the spinal cord in botulism].

作者信息

Chesnokova N P, Kuliash G Iu

出版信息

Biull Eksp Biol Med. 1985 Oct;100(10):442-4.

PMID:2996653
Abstract

The experiments on white rats have shown that gutimin is capable of reactivating Na, K-ATPase of the synaptosomes of the jugular spinal cord in type C botulinic intoxication. Serotonin prevented Na, K-ATPase activity inhibition only in preclinical period of intoxication. Parmidin injection did not prevent suppression of Na, K-ATPase activity either in preclinical period or in skeletal muscle paresis.

摘要

对白鼠的实验表明，古替明能够使C型肉毒中毒时颈脊髓突触体的钠钾-ATP酶重新激活。血清素仅在中毒的临床前期阻止了钠钾-ATP酶活性的抑制。帕尔米丁注射在临床前期或骨骼肌麻痹期均未阻止钠钾-ATP酶活性的抑制。

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