Macrophages: The Bridge between Inflammation Resolution and Tissue Repair?
作者信息
Garlet G P, Giannobile W V
机构信息
1 Department of Biological Sciences, School of Dentistry of Bauru, São Paulo University (FOB/USP), Bauru, SP, Brazil.
2 Department of Periodontics and Oral Medicine, University of Michigan School of Dentistry, Ann Arbor, MI, USA.
出版信息
J Dent Res. 2018 Sep;97(10):1079-1081. doi: 10.1177/0022034518785857. Epub 2018 Jul 11.
Abstract
摘要
相似文献
1
Macrophages: The Bridge between Inflammation Resolution and Tissue Repair?巨噬细胞：炎症消退与组织修复之间的桥梁？
J Dent Res. 2018 Sep;97(10):1079-1081. doi: 10.1177/0022034518785857. Epub 2018 Jul 11.
2
Wound healing in periodontal disease induces macrophage polarization characterized by different arginine-metabolizing enzymes.牙周病中的伤口愈合诱导具有不同精氨酸代谢酶特征的巨噬细胞极化。
J Periodontal Res. 2022 Apr;57(2):357-370. doi: 10.1111/jre.12965. Epub 2021 Dec 17.
3
The role of macrophages in the periodontal regeneration using Emdogain gel.巨噬细胞在使用Emdogain凝胶进行牙周再生中的作用。
J Periodontal Res. 2008 Apr;43(2):143-55. doi: 10.1111/j.1600-0765.2007.01004.x.
4
Macrophages in inflammation, repair and regeneration.炎症、修复和再生中的巨噬细胞。
Int Immunol. 2018 Oct 29;30(11):511-528. doi: 10.1093/intimm/dxy054.
5
Osteoimmunology: Inflammatory osteolysis and regeneration of the alveolar bone.骨免疫学：炎症性骨溶解与牙槽骨再生。
J Clin Periodontol. 2019 Jun;46 Suppl 21:52-69. doi: 10.1111/jcpe.13056.
6
Macrophages contribute to periodontal wound healing mainly in the tissue proliferation stage.巨噬细胞主要在组织增生期促进牙周伤口愈合。
J Periodontal Res. 2023 Feb;58(1):122-130. doi: 10.1111/jre.13074. Epub 2022 Nov 18.
7
Inflammation and Periodontal Regeneration.炎症与牙周组织再生
Dent Clin North Am. 2022 Jan;66(1):39-51. doi: 10.1016/j.cden.2021.08.003. Epub 2021 Oct 9.
8
Cytokines during periodontal wound healing: potential application for new therapeutic approach.牙周伤口愈合过程中的细胞因子：新治疗方法的潜在应用
Oral Dis. 2017 Apr;23(3):300-311. doi: 10.1111/odi.12469. Epub 2016 Mar 28.
9
Influence of parstatin on experimental periodontal disease and repair in rats.帕司他汀对大鼠实验性牙周病及修复的影响
J Periodontol. 2014 Sep;85(9):1266-74. doi: 10.1902/jop.2014.130619. Epub 2014 Jan 13.
10
Metabolic regulation of macrophages during tissue repair: insights from skeletal muscle regeneration.组织修复过程中巨噬细胞的代谢调控：来自骨骼肌再生的见解
FEBS Lett. 2017 Oct;591(19):3007-3021. doi: 10.1002/1873-3468.12703. Epub 2017 Jun 17.
引用本文的文献
1
Recombinant human bone morphogenetic protein-2 (rhBMP-2) induced macrophage biphasic polarization regulated by dexamethasone in vivo.重组人骨形态发生蛋白-2（rhBMP-2）在体内诱导巨噬细胞的双相极化受地塞米松调节。
Am J Transl Res. 2025 Jun 15;17(6):4159-4174. doi: 10.62347/UGHK3747. eCollection 2025.
2
M1 and M2 macrophages markers are alternately expressed during periapical lesion development.在根尖周病变发展过程中，M1和M2巨噬细胞标志物交替表达。
J Appl Oral Sci. 2025 Jun 27;33:e20240579. doi: 10.1590/1678-7757-2024-0579. eCollection 2025.
3
Regulation of immune microenvironments by polyetheretherketone surface topography for improving osseointegration.聚醚醚酮表面形貌对免疫微环境的调控以改善骨整合
J Nanobiotechnology. 2025 Mar 11;23(1):199. doi: 10.1186/s12951-025-03272-7.
4
The impact of the soluble epoxide hydrolase cascade on periodontal tissues.可溶性环氧化物水解酶级联反应对牙周组织的影响。
Front Dent Med. 2023 Feb 1;4:1129371. doi: 10.3389/fdmed.2023.1129371. eCollection 2023.
5
Unraveling the Applicability of LbL Coatings for Drug Delivery in Dental Implant-Related Infection Treatment.解析层层自组装涂层在牙种植体相关感染治疗中用于药物递送的适用性
ACS Biomater Sci Eng. 2025 Jan 13;11(1):13-32. doi: 10.1021/acsbiomaterials.4c01037. Epub 2024 Nov 30.
6
Dynamic changes in macrophage polarization during the resolution phase of periodontal disease.牙周病缓解期内巨噬细胞极化的动态变化。
Immun Inflamm Dis. 2024 Oct;12(10):e70044. doi: 10.1002/iid3.70044.
7
The role of WTAP in regulating macrophage-mediated osteoimmune responses and tissue regeneration in periodontitis.WTAP 在调节巨噬细胞介导的牙周炎骨免疫反应和组织再生中的作用。
Front Immunol. 2024 Jul 16;15:1423378. doi: 10.3389/fimmu.2024.1423378. eCollection 2024.
8
PGRN is involved in macrophage M2 polarization regulation through TNFR2 in periodontitis.PGRN 通过 TNFR2 参与牙周炎中巨噬细胞 M2 极化的调节。
J Transl Med. 2024 Apr 30;22(1):407. doi: 10.1186/s12967-024-05214-7.
9
Immune dysregulation and macrophage polarization in peri-implantitis.种植体周围炎中的免疫失调与巨噬细胞极化
Front Bioeng Biotechnol. 2024 Jan 29;12:1291880. doi: 10.3389/fbioe.2024.1291880. eCollection 2024.
10
Modulating the sEH/EETs Axis Restrains Specialized Proresolving Mediator Impairment and Regulates T Cell Imbalance in Experimental Periodontitis.调节 sEH/EETs 轴抑制特化促解决介质损伤并调节实验性牙周炎中的 T 细胞失衡。
J Immunol. 2024 Feb 1;212(3):433-445. doi: 10.4049/jimmunol.2300650.
本文引用的文献
1
Resolving Macrophages Counter Osteolysis by Anabolic Actions on Bone Cells.通过对成骨细胞的合成代谢作用来解决破骨细胞引发的骨吸收。
J Dent Res. 2018 Sep;97(10):1160-1169. doi: 10.1177/0022034518777973. Epub 2018 Jul 11.
2
Macrophage plasticity, polarization, and function in health and disease.巨噬细胞的可塑性、极化及其在健康与疾病中的功能。
J Cell Physiol. 2018 Sep;233(9):6425-6440. doi: 10.1002/jcp.26429. Epub 2018 Mar 1.
3
Macrophages in Tissue Repair, Regeneration, and Fibrosis.组织修复、再生和纤维化中的巨噬细胞
Immunity. 2016 Mar 15;44(3):450-462. doi: 10.1016/j.immuni.2016.02.015.
4
Alternatively Activated Macrophages Boost Induced Regulatory T and Th17 Cell Responses during Immunotherapy for Colitis.交替活化的巨噬细胞在结肠炎免疫治疗期间增强诱导性调节性T细胞和Th17细胞反应。
J Immunol. 2016 Apr 15;196(8):3305-17. doi: 10.4049/jimmunol.1501956. Epub 2016 Feb 29.
5
Characterization of the human immune cell network at the gingival barrier.牙龈屏障处人类免疫细胞网络的特征分析。
Mucosal Immunol. 2016 Sep;9(5):1163-1172. doi: 10.1038/mi.2015.136. Epub 2016 Jan 6.
6
Bone Engineering of Maxillary Sinus Bone Deficiencies Using Enriched CD90+ Stem Cell Therapy: A Randomized Clinical Trial.使用富集CD90+干细胞疗法对上颌窦骨缺损进行骨工程：一项随机临床试验。
J Bone Miner Res. 2015 Jul;30(7):1206-16. doi: 10.1002/jbmr.2464.
7
IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis.白细胞介素-4/趋化因子配体22/趋化因子受体4轴调控调节性T细胞迁移，从而抑制小鼠实验性牙周炎中的炎性骨质流失。
J Bone Miner Res. 2015 Mar;30(3):412-22. doi: 10.1002/jbmr.2376.
8
Pro-resolving lipid mediators are leads for resolution physiology.促炎消退脂质介质是解决生理学的研究线索。
Nature. 2014 Jun 5;510(7503):92-101. doi: 10.1038/nature13479.
9
Foxp3+ CD4+ T cells improve healing after myocardial infarction by modulating monocyte/macrophage differentiation.Foxp3+ CD4+ T 细胞通过调节单核细胞/巨噬细胞分化来改善心肌梗死后的愈合。
Circ Res. 2014 Jun 20;115(1):55-67. doi: 10.1161/CIRCRESAHA.115.303895. Epub 2014 Apr 30.
10
Macrophages: supportive cells for tissue repair and regeneration.巨噬细胞：组织修复和再生的支持细胞。
Immunobiology. 2014 Mar;219(3):172-8. doi: 10.1016/j.imbio.2013.09.001. Epub 2013 Sep 8.
Zotero 插件