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Carbon monoxide releasing molecule-3 inhibits inflammasome activation: A potential therapy for spinal cord injury.

作者信息

de Rivero Vaccari Juan Pablo

机构信息

Department of Neurological Surgery, Neuroscience Program, The Miami Project to Cure Paralysis, University of Miami Miller School of Medicine, Miami, FL, United States.

出版信息

EBioMedicine. 2019 Feb;40:17-18. doi: 10.1016/j.ebiom.2019.01.020. Epub 2019 Jan 14.

DOI:10.1016/j.ebiom.2019.01.020
PMID:30651220
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6413334/
Abstract
摘要

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本文引用的文献

1
Carbon monoxide releasing molecule-3 alleviates neuron death after spinal cord injury via inflammasome regulation.一氧化碳释放分子-3 通过炎症小体调节减轻脊髓损伤后的神经元死亡。
EBioMedicine. 2019 Feb;40:643-654. doi: 10.1016/j.ebiom.2018.12.059. Epub 2019 Jan 3.
2
IRE1α inhibition decreased TXNIP/NLRP3 inflammasome activation through miR-17-5p after neonatal hypoxic-ischemic brain injury in rats.IRE1α 抑制通过 miR-17-5p 降低大鼠新生缺氧缺血性脑损伤后的 TXNIP/NLRP3 炎性小体激活。
J Neuroinflammation. 2018 Feb 2;15(1):32. doi: 10.1186/s12974-018-1077-9.
3
Traumatic Spinal Cord Injury-Repair and Regeneration.创伤性脊髓损伤——修复与再生
Neurosurgery. 2017 Mar 1;80(3S):S9-S22. doi: 10.1093/neuros/nyw080.
4
Cleavage of GSDMD by inflammatory caspases determines pyroptotic cell death.炎性小体天冬氨酸特异性半胱氨酸蛋白酶 1（caspase-1）切割 GSDMD 决定细胞焦亡。
Nature. 2015 Oct 29;526(7575):660-5. doi: 10.1038/nature15514. Epub 2015 Sep 16.
5
Activation and regulation of cellular inflammasomes: gaps in our knowledge for central nervous system injury.细胞焦亡体的激活与调控：中枢神经系统损伤相关研究中的知识缺口。
J Cereb Blood Flow Metab. 2014 Mar;34(3):369-75. doi: 10.1038/jcbfm.2013.227. Epub 2014 Jan 8.
6
Pyroptotic neuronal cell death mediated by the AIM2 inflammasome.AIM2 炎性小体介导的细胞焦亡性神经元死亡。
J Cereb Blood Flow Metab. 2014 Apr;34(4):621-9. doi: 10.1038/jcbfm.2013.236. Epub 2014 Jan 8.
7
From basics to clinical: a comprehensive review on spinal cord injury.从基础到临床：脊髓损伤的全面综述。
Prog Neurobiol. 2014 Mar;114:25-57. doi: 10.1016/j.pneurobio.2013.11.002. Epub 2013 Nov 20.
8
The therapeutic potential of carbon monoxide.一氧化碳的治疗潜力。
Nat Rev Drug Discov. 2010 Sep;9(9):728-43. doi: 10.1038/nrd3228.
9
A molecular platform in neurons regulates inflammation after spinal cord injury.神经元中的一个分子平台可调节脊髓损伤后的炎症反应。
J Neurosci. 2008 Mar 26;28(13):3404-14. doi: 10.1523/JNEUROSCI.0157-08.2008.
10
The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta.炎性小体：触发炎性半胱天冬酶激活和前白细胞介素-β加工的分子平台。
Mol Cell. 2002 Aug;10(2):417-26. doi: 10.1016/s1097-2765(02)00599-3.