致敏脾细胞和抗体对柯萨奇病毒B4感染的体外和体内传播的控制 - Suppr

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超能文献

Control of in vitro and in vivo spread of coxsackievirus B4 infection by sensitized spleen cells and antibody.

作者信息

Chaturvedi U C, Tandon H O, Mathur A

出版信息

J Infect Dis. 1978 Aug;138(2):181-90. doi: 10.1093/infdis/138.2.181.

DOI:10.1093/infdis/138.2.181

PMID:308084

Abstract

摘要

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Control of in vitro and in vivo spread of coxsackievirus B4 infection by sensitized spleen cells and antibody.致敏脾细胞和抗体对柯萨奇病毒B4感染的体外和体内传播的控制

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引用本文的文献

Binding of macrophages and B lymphocytes mediated by dengue virus antigen and the virus-induced helper cytokine.由登革病毒抗原和病毒诱导的辅助细胞因子介导的巨噬细胞与B淋巴细胞的结合。

Int J Exp Pathol. 1993 Apr;74(2):187-94.

Specific receptors for dengue virus-induced suppressor cytokine on macrophages and lymphocytes.登革病毒诱导的抑制性细胞因子在巨噬细胞和淋巴细胞上的特异性受体。

Int J Exp Pathol. 1994 Feb;75(1):29-36.

Characterization of the cytotoxic factor produced in the spleen of dengue virus-infected mice.登革病毒感染小鼠脾脏中产生的细胞毒性因子的特性分析。

Immunology. 1980 Oct;41(2):387-92.

Depressed macrophage functions in dengue virus-infected mice: role of the cytotoxic factor.登革病毒感染小鼠中巨噬细胞功能的抑制：细胞毒性因子的作用

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Dengue virus-induced cytotoxic factor suppresses immune response of mice to sheep erythrocytes.登革病毒诱导的细胞毒性因子抑制小鼠对绵羊红细胞的免疫反应。

Immunology. 1981 Jun;43(2):311-6.

Inhibition of E-rosette formation and phagocytosis by human blood leucocytes after treatment with the dengue virus-induced cytotoxic factor.登革病毒诱导的细胞毒性因子处理后对人血白细胞E-玫瑰花结形成和吞噬作用的抑制

Immunology. 1982 Apr;45(4):679-85.

Production of cytotoxic factor in the spleen of dengue virus-infected mice.登革病毒感染小鼠脾脏中细胞毒性因子的产生

Immunology. 1980 Aug;40(4):665-71.

Dengue virus-induced cytotoxic factor induces macrophages to produce a cytotoxin.登革病毒诱导的细胞毒性因子可诱导巨噬细胞产生一种细胞毒素。

Immunology. 1983 May;49(1):121-30.

Thymus-dependent lymphocytes of dengue virus-infected mice spleens mediate suppression through prostaglandin.登革病毒感染小鼠脾脏中的胸腺依赖性淋巴细胞通过前列腺素介导抑制作用。

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In vivo role of macrophages in transmission of dengue virus-induced suppressor signal to T lymphocytes.巨噬细胞在登革病毒诱导的抑制信号向T淋巴细胞传递中的体内作用。

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