Department of Psychology, Faculty of Arts, Psychology and Theology, Åbo Akademi University.
Department of Psychology and Speech-Language Pathology, University of Turku.
J Clin Child Adolesc Psychol. 2022 Jul-Aug;51(4):505-514. doi: 10.1080/15374416.2020.1731820. Epub 2020 Mar 16.
Bullying affects approximately a quarter of schoolchildren and is associated with numerous adverse outcomes. Although distinct risk factors for bullying and victimization have been identified, few studies have investigated the genetic and environmental underpinnings of bullying and victimization. The aims of this study were twofold: first, to examine the contributions of genetic and environmental factors to bullying and victimization, and second, to analyze whether the KiVa antibullying program moderated the magnitude of these contributions by comparing estimates derived from the KiVa versus control groups.
The sample comprised students from schools that participated in the evaluation of the KiVa antibullying program in Finland during 2007-2009. Bullying and victimization were measured using peer nominations by classmates. The sample for the twin analyses comprised of 447 twins (107 monozygotic and 340 dizygotic twins) aged 7-15.
Genetic contributions accounted for 62% and 77% of the variance in bullying and in victimization at pre-intervention, respectively. There was a post-intervention difference in the overall role of genetic and environmental contributions between the intervention and the control group for bullying and victimization, with non-shared environmental effects playing a lesser role (and genes a larger role) in the intervention than in the control group context.
This study replicates previous findings on the genetic underpinnings of both bullying and victimization, and indicates that a school-based antibullying program reduces the role of non-shared environmental factors in bullying and victimization. The results indicate that prevention and intervention efforts need to target both environmental and (heritable) individual level factors to maximize effectiveness.
大约四分之一的学童受到欺凌的影响,且欺凌与许多不良后果相关。尽管已确定欺凌和受害的独特风险因素,但很少有研究调查欺凌和受害的遗传和环境基础。本研究的目的有两个:首先,检验遗传和环境因素对欺凌和受害的贡献;其次,通过比较 KiVa 与对照组得出的估计值,分析 KiVa 反欺凌计划是否会调节这些贡献的大小。
该样本包括参加 2007-2009 年在芬兰进行的 KiVa 反欺凌计划评估的学校的学生。使用同学提名来衡量欺凌和受害情况。双胞胎分析的样本包括 447 对双胞胎(107 对同卵双胞胎和 340 对异卵双胞胎),年龄在 7-15 岁之间。
遗传因素分别解释了干预前欺凌和受害的 62%和 77%的方差。在干预和对照组之间,干预后遗传和环境因素对欺凌和受害的总体作用存在差异,非共享环境效应在干预组中的作用较小(而基因的作用较大),而在对照组中则相反。
本研究复制了之前关于欺凌和受害遗传基础的发现,并表明基于学校的反欺凌计划减少了非共享环境因素在欺凌和受害中的作用。研究结果表明,预防和干预措施需要针对环境和(可遗传的)个体水平因素,以最大限度地提高效果。