How NETosis could drive "Post-COVID-19 syndrome" among survivors.
作者信息
Sawadogo Serge A, Dighero-Kemp Bonnie, Ouédraogo Dieu-Donné, Hensley Lisa, Sakandé Jean
机构信息
Université Joseph Ki Zerbo, Unité de Formation et de Recherche en Sciences de la Santé, 03 BP 7021 Ouagadougou 03, Burkina Faso; Institut pour la Promotion de la Recherche et de l'Innovation en Immunologie Médicale de Ouagadougou (PrïmO) site Nelson Mandela, Immunology department, 09 BP 706, Ouagadougou 09, Burkina Faso.
National Institute of Allergy and Infectious Diseases (NIAID) Integrated Research Facility Division of Clinical Research, Virology Department, 8200 Research Plaza Fort Detrick, MD, 21702, United States.
出版信息
Immunol Lett. 2020 Dec;228:35-37. doi: 10.1016/j.imlet.2020.09.005. Epub 2020 Sep 29.
Abstract
摘要
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3
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