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Vitamin D supplementation improves simvastatin-mediated decline in exercise performance: A randomized double-blind placebo-controlled study.维生素 D 补充可改善辛伐他汀介导的运动表现下降:一项随机、双盲、安慰剂对照研究。
J Diabetes. 2017 Dec;9(12):1100-1106. doi: 10.1111/1753-0407.12541. Epub 2017 Jun 28.
4
Mitochondrial disease: genetics and management.线粒体疾病:遗传学与管理
J Neurol. 2016 Jan;263(1):179-91. doi: 10.1007/s00415-015-7884-3. Epub 2015 Aug 28.
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Bisphenol A induces oxidative stress and mitochondrial dysfunction in lymphoblasts from children with autism and unaffected siblings.双酚A可诱导自闭症儿童及其未受影响的同胞淋巴细胞中的氧化应激和线粒体功能障碍。
Free Radic Biol Med. 2014 Nov;76:25-33. doi: 10.1016/j.freeradbiomed.2014.07.030. Epub 2014 Aug 4.
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Mono-(2-ethylhexyl) phthalate induces injury in human umbilical vein endothelial cells.邻苯二甲酸单(2-乙基己基)酯诱导人脐静脉内皮细胞损伤。
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Toxicity of Arsenic (III) on Isolated Liver Mitochondria: A New Mechanistic Approach.三价砷对离体肝线粒体的毒性:一种新的机制研究方法。
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保护线粒体免受金属和化学物质影响的策略

Strategies for Protecting Mitochondria From Metals and Chemicals.

作者信息

Pizzorno Joseph

出版信息

Integr Med (Encinitas). 2022 May;21(2):8-13.

PMID:35698610
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9173845/
Abstract

Mitochondria are required for life, and dysfunction leads to chronic disease and shortened life expectancy. Unfortunately, suboptimal mitochondrial function is very common. While genetic mutations play a role, far more common is the damage from nutrient deficiencies and regular accidental and intentional exposure to mitotoxic metals and chemicals. Alcohol, antibiotics, metals, bisphenols, phthalates, pesticides and herbicides, and statins-the list is long-all impair mitochondrial function. Fortunately, toxins can be avoided and their elimination from the body can be enhanced. In addition, many natural health molecules help protect mitochondria and restore function.

摘要

线粒体是生命所必需的,其功能障碍会导致慢性疾病并缩短预期寿命。不幸的是,线粒体功能欠佳的情况非常普遍。虽然基因突变会起一定作用,但更常见的是营养缺乏以及经常意外和有意接触线粒体毒性金属和化学物质所造成的损害。酒精、抗生素、金属、双酚、邻苯二甲酸盐、杀虫剂和除草剂以及他汀类药物——不胜枚举——都会损害线粒体功能。幸运的是,可以避免接触毒素,还能加强从体内清除毒素。此外,许多天然健康分子有助于保护线粒体并恢复其功能。