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2002年至2022年脓毒症诱导性心肌功能障碍的文献计量分析

A bibliometric analysis of sepsis-induced myocardial dysfunction from 2002 to 2022.

作者信息

Yao Hanyi, Liu Shufang, Zhang Zhiyu, Xiao Zixi, Li Dongping, Yi Zhangqing, Huang Yuyang, Zhou Haojie, Yang Yifeng, Zhang Weizhi

机构信息

Department of Cardiovascular Surgery, The Second Xiangya Hospital, Central South University, Changsha, China.

Clinical Center for Gene Diagnosis and Therapy, The Second Xiangya Hospital, Central South University, Changsha, China.

出版信息

Front Cardiovasc Med. 2023 Jan 30;10:1076093. doi: 10.3389/fcvm.2023.1076093. eCollection 2023.

DOI:10.3389/fcvm.2023.1076093
PMID:36793476
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9922860/
Abstract

BACKGROUND

Sepsis-induced myocardial dysfunction (SIMD) has a significant contribution to sepsis-caused death in critically ill patients. In recent years, the number of published articles related to SIMD has increased rapidly. However, there was no literature that systematically analyzed and evaluated these documents. Thus, we aimed to lay a foundation for researchers to quickly understand the research hotspots, evolution processes and development trends in the SIMD field a bibliometric analysis.

METHODS

Articles related to SIMD were retrieved and extracted from the Web of Science Core Collection on July 19th, 2022. CiteSpace (version 6.1.R2) and VOSviewer (version 1.6.18) were used for performing visual analysis.

RESULTS

A total of 1,076 articles were included. The number of SIMD-related articles published each year has increased significantly. These publications mainly came from 56 countries, led by China and the USA, and 461 institutions, but without stable and close cooperation. As authors, Li Chuanfu published the most articles, while Rudiger Alain had the most co-citations. Shock was the journal with the most studies, and Critical Care Medicine was the most commonly cited journal. All keywords were grouped into six clusters, some of which represented the current and developing research directions of SIMD as the molecular mechanisms.

CONCLUSION

Research on SIMD is flourishing. It is necessary to strengthen cooperation and exchanges between countries and institutions. The molecular mechanisms of SIMD, especially oxidative stress and regulated cell death, will be critical subjects in the future.

摘要

背景

脓毒症诱导的心肌功能障碍(SIMD)对危重症患者因脓毒症导致的死亡有重大影响。近年来,与SIMD相关的已发表文章数量迅速增加。然而,尚无文献对这些文献进行系统的分析和评估。因此,我们旨在通过文献计量分析为研究人员快速了解SIMD领域的研究热点、演变过程和发展趋势奠定基础。

方法

于2022年7月19日从科学网核心合集检索并提取与SIMD相关的文章。使用CiteSpace(6.1.R2版本)和VOSviewer(1.6.18版本)进行可视化分析。

结果

共纳入1076篇文章。每年发表的与SIMD相关的文章数量显著增加。这些出版物主要来自56个国家,以中国和美国为首,以及461个机构,但缺乏稳定且紧密的合作。作为作者,李传福发表的文章最多,而鲁迪格·阿兰的共被引次数最多。《休克》是研究最多的期刊,《危重病医学》是最常被引用的期刊。所有关键词被分为六个聚类,其中一些代表了SIMD当前和正在发展的研究方向,如分子机制。

结论

对SIMD的研究正在蓬勃发展。有必要加强国家和机构之间的合作与交流。SIMD的分子机制,尤其是氧化应激和程序性细胞死亡,将是未来的关键课题。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/a498a60e8fa8/fcvm-10-1076093-g009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/e3c8cd3b3dc3/fcvm-10-1076093-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/b9bff6f921b5/fcvm-10-1076093-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/a61ee7b988c5/fcvm-10-1076093-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/c0d8f3b0cb03/fcvm-10-1076093-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/9a5c3227aee8/fcvm-10-1076093-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/19e3486438c5/fcvm-10-1076093-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/f911319ea4ea/fcvm-10-1076093-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/37c3eacca7c1/fcvm-10-1076093-g008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/a498a60e8fa8/fcvm-10-1076093-g009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/e3c8cd3b3dc3/fcvm-10-1076093-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/b9bff6f921b5/fcvm-10-1076093-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/a61ee7b988c5/fcvm-10-1076093-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/c0d8f3b0cb03/fcvm-10-1076093-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/9a5c3227aee8/fcvm-10-1076093-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/19e3486438c5/fcvm-10-1076093-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/f911319ea4ea/fcvm-10-1076093-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/37c3eacca7c1/fcvm-10-1076093-g008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0d73/9922860/a498a60e8fa8/fcvm-10-1076093-g009.jpg

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