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内质网应激与动脉粥样硬化的文献计量分析

A bibliometric analysis of endoplasmic reticulum stress and atherosclerosis.

作者信息

Huang Xinyu, Jiang Feng, Ma Yongbo, Zhu Kunpeng, Wang Zhenyuan, Hua Zhen, Yu Jie, Zhang Lei

机构信息

Shandong University of Traditional Chinese Medicine, Shandon, China.

Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Shandon, China.

出版信息

Front Physiol. 2024 Jun 11;15:1392454. doi: 10.3389/fphys.2024.1392454. eCollection 2024.

DOI:10.3389/fphys.2024.1392454
PMID:38938744
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11210825/
Abstract

The mechanisms underlying the occurrence and development of atherosclerosis (AS) are diverse, among which endoplasmic reticulum stress (ERS) is an important mechanism that should not be overlooked. However, up to now, there has been no bibliometric study on the relationship between ERS and AS. To understand the research progress in ERS and AS, this paper conducted a statistical analysis of publications in this field using bibliometrics. A total of 1,035 records were retrieved from the Web of Science Core Collection. CiteSpace, VOSviewer, and the R package "bibliometric" were used to analyze the spatiotemporal distribution, countries, authors, institutions, journals, references, and keywords of the literature, and to present the basic information of this field through visualized maps, as well as determine the collaboration relationships among researchers in this field. This field has gradually developed and stabilized over the past 20 years. The current research hotspots in this field mainly include the relationship between ERS and AS-related cells, the mechanisms by which ERS promotes AS, related diseases, and associated cytokines, etc. Vascular calcification, endothelial dysfunction, NLRP3 inflammasome, and heart failure represent the frontier research in this field and are becoming new research hotspots. It is hoped that this study will provide new insights for research and clinical work in the field of ERS and AS.

摘要

动脉粥样硬化（AS）发生和发展的潜在机制是多样的，其中内质网应激（ERS）是一个不可忽视的重要机制。然而，截至目前，尚未有关于ERS与AS关系的文献计量学研究。为了解ERS和AS的研究进展，本文运用文献计量学方法对该领域的出版物进行了统计分析。从科学网核心合集共检索到1035条记录。使用CiteSpace、VOSviewer和R包“bibliometric”对文献的时空分布、国家、作者、机构、期刊、参考文献和关键词进行分析，并通过可视化地图展示该领域的基本信息，以及确定该领域研究人员之间的合作关系。在过去20年中，该领域逐渐发展并趋于稳定。该领域当前的研究热点主要包括ERS与AS相关细胞的关系、ERS促进AS的机制、相关疾病以及相关细胞因子等。血管钙化、内皮功能障碍、NLRP3炎性小体和心力衰竭代表了该领域的前沿研究，正成为新的研究热点。希望本研究能为ERS和AS领域的研究及临床工作提供新的见解。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/8d0d96a58f9d/fphys-15-1392454-g009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/a6317bf4c0ff/fphys-15-1392454-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/0fdcb9bc5dbe/fphys-15-1392454-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/67963d24b2a1/fphys-15-1392454-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/8b6ede29a79f/fphys-15-1392454-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/10354d6bc1be/fphys-15-1392454-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/7a2c1751d4d2/fphys-15-1392454-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/c73fed1d282e/fphys-15-1392454-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/764ba55eaaf0/fphys-15-1392454-g008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/8d0d96a58f9d/fphys-15-1392454-g009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/a6317bf4c0ff/fphys-15-1392454-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/0fdcb9bc5dbe/fphys-15-1392454-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/67963d24b2a1/fphys-15-1392454-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/8b6ede29a79f/fphys-15-1392454-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/10354d6bc1be/fphys-15-1392454-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/7a2c1751d4d2/fphys-15-1392454-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/c73fed1d282e/fphys-15-1392454-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/764ba55eaaf0/fphys-15-1392454-g008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a517/11210825/8d0d96a58f9d/fphys-15-1392454-g009.jpg

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本文引用的文献

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