Department of Trauma, Hand and Reconstructive Surgery, University Hospital Münster, Münster, Germany.
OCM Clinic, Munich, Germany.
Am J Sports Med. 2024 Nov;52(13):3306-3313. doi: 10.1177/03635465241280984. Epub 2024 Oct 3.
Injuries of the superficial medial collateral ligament (sMCL) and anteromedial structures of the knee result in excess valgus rotation and external tibial rotation (ER) as well as tibial translation.
To evaluate a flat reconstruction of the sMCL and anteromedial structures in restoring knee kinematics in the combined MCL- and anteromedial-deficient knee.
Controlled laboratory study.
Eight cadaveric knee specimens were tested in a 6 degrees of freedom robotic test setup. Force-controlled clinical laxity tests were performed with 200 N of axial compression in 0°, 30°, 60°, and 90° of flexion: 8 N·m valgus torque, 5 N·m ER torque, 89 N anterior tibial translation (ATT) force, and an anteromedial drawer test consisting of 89 N ATT force under 5 N·m ER torque. After determining the native knee kinematics, we transected the sMCL, followed by the deep medial collateral ligament (dMCL). Subsequently, a flat reconstruction of the sMCL with anteromedial limb, mimicking the function of the anteromedial corner, was performed. Mixed linear models were used for statistical analysis ( < .05).
Cutting of the sMCL led to statistically significant increases in laxity regarding valgus rotation, ER, and anteromedial translation in all tested flexion angles ( < .05). ATT was significantly increased in all flexion angles but not at 60° after cutting of the sMCL. A combined instability of the sMCL and dMCL led to further increased knee laxity in all tested kinematics and flexion angles ( < .05). After reconstruction, the knee kinematics were not significantly different from those of the native state.
Insufficiency of the sMCL and dMCL led to excess valgus rotation, ER, ATT, and anteromedial tibial translation. A combined flat reconstruction of the sMCL and the anteromedial aspect restored this excess laxity to values not significantly different from those of the native knee.
The presented reconstruction might lead to favorable results for patients with MCL and anteromedial injuries with an anteromedial rotatory knee instability.
膝关节内侧副韧带浅层(sMCL)和前内侧结构的损伤可导致过度外翻旋转和胫骨外旋(ER)以及胫骨平移。
评估 sMCL 及其前内侧结构的平面重建在重建 MCL 和前内侧结构同时缺失的膝关节的膝关节运动学中的作用。
对照实验室研究。
在 6 自由度机器人测试设置中测试了 8 个尸体膝关节标本。在 0°、30°、60°和 90°的屈曲下进行力控制的临床松弛度测试:8 N·m 外翻扭矩、5 N·m ER 扭矩、89 N 胫骨前平移(ATT)力和前内侧抽屉测试(在 5 N·m ER 扭矩下施加 89 N ATT 力)。在确定自然膝关节运动学后,我们切断 sMCL,然后切断深内侧副韧带(dMCL)。随后,进行 sMCL 及其前内侧支的平面重建,模拟前内侧角的功能。采用混合线性模型进行统计学分析( <.05)。
切断 sMCL 导致在所有测试的屈曲角度下,外翻旋转、ER 和前内侧平移的松弛度均有统计学显著增加( <.05)。在切断 sMCL 后,在所有屈曲角度下 ATT 均显著增加,但在 60°时除外。sMCL 和 dMCL 的联合不稳定性导致在所有测试的运动学和屈曲角度下膝关节松弛度进一步增加( <.05)。重建后,膝关节运动学与自然状态无显著差异。
sMCL 和 dMCL 的不足导致过度外翻旋转、ER、ATT 和前内侧胫骨平移。sMCL 和前内侧结构的联合平面重建将这种过度松弛度恢复到与自然膝关节无显著差异的值。
对于 MCL 和前内侧损伤伴前内侧旋转不稳定的患者,所提出的重建可能会带来良好的效果。