Immune-enhancing effects of endogenous glucocorticoids on gastric macrophages contribute to the development of gastric inflammation and metaplasia.
作者信息
Bimczok Diane
机构信息
Department of Microbiology and Cell BiologyMontana State University, Bozeman, Montana, United States.
出版信息
Am J Physiol Gastrointest Liver Physiol. 2024 Dec 1;327(6):G874-G876. doi: 10.1152/ajpgi.00299.2024. Epub 2024 Nov 5.
Abstract
摘要
相似文献
1
Immune-enhancing effects of endogenous glucocorticoids on gastric macrophages contribute to the development of gastric inflammation and metaplasia.内源性糖皮质激素对胃巨噬细胞的免疫增强作用促进了胃炎症和化生的发展。
Am J Physiol Gastrointest Liver Physiol. 2024 Dec 1;327(6):G874-G876. doi: 10.1152/ajpgi.00299.2024. Epub 2024 Nov 5.
2
Endogenous glucocorticoids are required for normal macrophage activation and gastric immunity.内源性糖皮质激素是正常巨噬细胞激活和胃免疫所必需的。
Am J Physiol Gastrointest Liver Physiol. 2024 Oct 1;327(4):G531-G544. doi: 10.1152/ajpgi.00114.2024. Epub 2024 Jul 23.
3
Glucocorticoids and Androgens Protect From Gastric Metaplasia by Suppressing Group 2 Innate Lymphoid Cell Activation.糖皮质激素和雄激素通过抑制 2 类固有淋巴细胞的激活来防止胃化生。
Gastroenterology. 2021 Aug;161(2):637-652.e4. doi: 10.1053/j.gastro.2021.04.075. Epub 2021 May 7.
4
Endogenous glucocorticoids prevent gastric metaplasia by suppressing spontaneous inflammation.内源性糖皮质激素通过抑制自发性炎症预防胃化生。
J Clin Invest. 2019 Mar 1;129(3):1345-1358. doi: 10.1172/JCI123233. Epub 2019 Feb 18.
5
Endogenous glucocorticoids are required for normal macrophage activation and gastric immunity.内源性糖皮质激素是正常巨噬细胞激活和胃免疫所必需的。
bioRxiv. 2024 Jan 15:2024.01.14.575574. doi: 10.1101/2024.01.14.575574.
6
Comparison of gastric inflammation and metaplasia induced by or colonization in mice.比较 或 定植诱导小鼠胃炎症和化生的情况。
Microbiol Spectr. 2024 Jun 4;12(6):e0001524. doi: 10.1128/spectrum.00015-24. Epub 2024 Apr 29.
7
Deoxycholic acid-stimulated macrophage-derived exosomes promote spasmolytic polypeptide-expressing metaplasia in the stomach.去氧胆酸刺激巨噬细胞衍生的外泌体促进胃中收缩多肽表达的化生。
Biochem Biophys Res Commun. 2020 Apr 9;524(3):649-655. doi: 10.1016/j.bbrc.2020.01.159. Epub 2020 Feb 4.
8
Cyclooxigenase-2 and osteopontin in gastric pre-neoplastic lesions in relation to H-pylori infection and grade of inflammation.环氧化酶-2 和骨桥蛋白与 H.pylori 感染和炎症程度在胃前肿瘤病变中的关系。
Rev Esp Patol. 2020 Apr-Jun;53(2):79-87. doi: 10.1016/j.patol.2019.06.005. Epub 2019 Aug 28.
9
Macrophages promote progression of spasmolytic polypeptide-expressing metaplasia after acute loss of parietal cells.巨噬细胞促进壁细胞急性缺失后舒血管肠肽表达化生的进展。
Gastroenterology. 2014 Jun;146(7):1727-38.e8. doi: 10.1053/j.gastro.2014.02.007. Epub 2014 Feb 15.
10
Gastric foveolar metaplasia and gastric heterotopia in the duodenum: no evidence of an etiologic role for Helicobacter pylori.胃小凹上皮化生和十二指肠胃异位：幽门螺杆菌无病因学作用证据。
Hum Pathol. 2010 Nov;41(11):1593-600. doi: 10.1016/j.humpath.2010.04.010. Epub 2010 Jul 24.
本文引用的文献
1
Macrophage biology in the pathogenesis of infection.感染发病机制中的巨噬细胞生物学
Crit Rev Microbiol. 2025 May;51(3):399-416. doi: 10.1080/1040841X.2024.2366944. Epub 2024 Jul 31.
2
Endogenous glucocorticoids are required for normal macrophage activation and gastric immunity.内源性糖皮质激素是正常巨噬细胞激活和胃免疫所必需的。
Am J Physiol Gastrointest Liver Physiol. 2024 Oct 1;327(4):G531-G544. doi: 10.1152/ajpgi.00114.2024. Epub 2024 Jul 23.
3
Glucocorticoids and Androgens Protect From Gastric Metaplasia by Suppressing Group 2 Innate Lymphoid Cell Activation.糖皮质激素和雄激素通过抑制 2 类固有淋巴细胞的激活来防止胃化生。
Gastroenterology. 2021 Aug;161(2):637-652.e4. doi: 10.1053/j.gastro.2021.04.075. Epub 2021 May 7.
4
Endogenous glucocorticoids prevent gastric metaplasia by suppressing spontaneous inflammation.内源性糖皮质激素通过抑制自发性炎症预防胃化生。
J Clin Invest. 2019 Mar 1;129(3):1345-1358. doi: 10.1172/JCI123233. Epub 2019 Feb 18.
5
Immune regulation by glucocorticoids.糖皮质激素的免疫调节作用。
Nat Rev Immunol. 2017 Apr;17(4):233-247. doi: 10.1038/nri.2017.1. Epub 2017 Feb 13.
6
CagY Is an Immune-Sensitive Regulator of the Helicobacter pylori Type IV Secretion System.CagY是幽门螺杆菌IV型分泌系统的免疫敏感调节因子。
Gastroenterology. 2016 Dec;151(6):1164-1175.e3. doi: 10.1053/j.gastro.2016.08.014. Epub 2016 Aug 26.
7
Pathobiology of Helicobacter pylori-Induced Gastric Cancer.幽门螺杆菌诱导的胃癌发病机制
Gastroenterology. 2016 Jan;150(1):64-78. doi: 10.1053/j.gastro.2015.09.004. Epub 2015 Sep 16.
8
Chronic exposure to glucocorticoids shapes gene expression and modulates innate and adaptive activation pathways in macrophages with distinct changes in leukocyte attraction.慢性暴露于糖皮质激素会影响巨噬细胞的基因表达，并调节先天和适应性激活途径，导致白细胞吸引的明显变化。
J Immunol. 2014 Feb 1;192(3):1196-208. doi: 10.4049/jimmunol.1302138. Epub 2014 Jan 6.
9
Glucocorticoid receptor dimerization is required for survival in septic shock via suppression of interleukin-1 in macrophages.糖皮质激素受体二聚化通过抑制巨噬细胞中的白细胞介素-1 从而在感染性休克中存活是必需的。
FASEB J. 2012 Feb;26(2):722-9. doi: 10.1096/fj.11-192112. Epub 2011 Oct 31.
10
Comparative whole genome sequence analysis of the carcinogenic bacterial model pathogen Helicobacter felis.致癌性细菌模型病原体弯曲杆菌的比较全基因组序列分析。
Genome Biol Evol. 2011;3:302-8. doi: 10.1093/gbe/evr022. Epub 2011 Mar 14.
Zotero 插件