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Immunofluorescence of green monkey kidney cells infected with adenovirus 12 and with adenovirus 12 plus simian virus 40.感染腺病毒12以及感染腺病毒12加猴病毒40的绿猴肾细胞的免疫荧光。
J Bacteriol. 1966 Jan;91(1):262-5. doi: 10.1128/jb.91.1.262-265.1966.
2
Influence of SV40 Genome on the Replication of an Adenovirus-SV40 "Hybrid" Population.猴空泡病毒40基因组对腺病毒-猴空泡病毒40“杂交”群体复制的影响
J Bacteriol. 1965 Sep;90(3):778-82. doi: 10.1128/jb.90.3.778-782.1965.
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Association of 20-Millimicron Particles with Adenoviruses.20 毫微米颗粒与腺病毒的关联
J Bacteriol. 1965 Jul;90(1):271-4. doi: 10.1128/jb.90.1.271-274.1965.
4
ADENOVIRUS-SV40 "HYBRIDS": PLAQUE PURIFICATION INTO LINES IN WHICH THE DETERMINANT FOR THE SV40 TUMOR ANTIGEN IS LOST OR RETAINED.腺病毒 - 猴病毒40“杂种病毒”：通过蚀斑纯化形成株系，其中猴病毒40肿瘤抗原的决定簇丢失或保留。
Virology. 1965 Jul;26:511-2. doi: 10.1016/0042-6822(65)90016-4.
5
DIFFERENTIAL EFFECTS OF INHIBITORS ON THE STEPS LEADING TO THE FORMATION OF SV40 TUMOR AND VIRUS ANTIGENS.抑制剂对导致SV40肿瘤和病毒抗原形成的各个步骤的不同作用。
J Exp Med. 1965 Jun 1;121(6):935-44. doi: 10.1084/jem.121.6.935.
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EARLY CHANGES IN PYRIMIDINE BIOSYNTHESIS AFTER PARTIAL HEPATECTOMY.肝部分切除术后嘧啶生物合成的早期变化
J Biol Chem. 1965 Jun;240:2550-6.
7
THE INCORPORATION OF SV40 MATERIAL INTO ADENOVIRUS 7 AS MEASURED BY INTRANUCLEAR SYNTHESIS OF SV40 TUMOR ANTIGEN.通过SV40肿瘤抗原的核内合成来测定SV40物质掺入腺病毒7的情况。
Proc Natl Acad Sci U S A. 1964 Dec;52(6):1348-52. doi: 10.1073/pnas.52.6.1348.
8
EVIDENCE FOR A POSSIBLE GENETIC HYBRID BETWEEN ADENOVIRUS TYPE 7 AND SV40 VIRUSES.7型腺病毒与猿猴空泡病毒40（SV40）之间可能存在基因杂交的证据。
Proc Natl Acad Sci U S A. 1964 Dec;52(6):1340-7. doi: 10.1073/pnas.52.6.1340.
9
INDUCTION BY ADENOVIRUS TYPE 7 OF TUMORS IN HAMSTERS HAVING THE ANTIGENIC CHARACTERISTICS OF SV40 VIRUS.7型腺病毒对具有SV40病毒抗原特性的仓鼠肿瘤的诱导作用
Proc Natl Acad Sci U S A. 1964 Dec;52(6):1333-40. doi: 10.1073/pnas.52.6.1333.
10
SYNTHESIS OF SV40 TUMOR ANTIGEN DURING REPLICATION OF SIMIAN PAPOVAVIRUS (SV40).猴乳头瘤空泡病毒（SV40）复制过程中SV40肿瘤抗原的合成
Proc Natl Acad Sci U S A. 1964 Nov;52(5):1138-42. doi: 10.1073/pnas.52.5.1138.

携带缺陷型猴细胞适应成分的7型腺病毒毒株的特性分析

Characterization of the strain of adenovirus type 7 carrying the defective monkey cell-adapting component.

作者信息

Butel J S

出版信息

J Virol. 1967 Oct;1(5):876-82. doi: 10.1128/JVI.1.5.876-882.1967.

DOI:10.1128/JVI.1.5.876-882.1967
PMID:4316236
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC375364/
Abstract

The strain of adenovirus type 7 carrying the defective monkey cell-adapting component (MAC) has been further characterized. MAC is more sensitive to inactivation by ultraviolet light than the associated adenovirus, which, in turn, is more rapidly inactivated than complete simian virus 40 (SV40). The 37% dose was 16 sec for MAC, 60 sec for adenovirus, and 84 sec for SV40. Filtration through membranes revealed that both MAC and adenovirus were retained by 100-mmu filters. MAC equilibrated in cesium chloride at the same buoyant density as the complete adenovirions (1.34 g/ml). Deoxyribonucleic acid extracted from green monkey kidney cells infected with MAC-adenovirus 7 did not hybridize in vitro with SV40 complementary ribonucleic acid. The two components (MAC and adenovirus) of the virus population possess such similar biophysical properties that their separation has not yet been achieved. Evidence to date indicates that MAC is probably not derived from SV40.

摘要

携带缺陷型猴细胞适应成分（MAC）的7型腺病毒毒株已得到进一步鉴定。与相关腺病毒相比，MAC对紫外线灭活更敏感，而腺病毒又比完整的猿猴病毒40（SV40）更快被灭活。MAC的37%剂量灭活时间为16秒，腺病毒为60秒，SV40为84秒。通过膜过滤显示，MAC和腺病毒均被100微米过滤器截留。MAC在氯化铯中平衡时的浮力密度与完整腺病毒颗粒相同（1.34克/毫升）。从感染MAC-腺病毒7的绿猴肾细胞中提取的脱氧核糖核酸在体外与SV40互补核糖核酸不杂交。病毒群体的两个成分（MAC和腺病毒）具有如此相似的生物物理特性，以至于尚未实现它们的分离。迄今为止的证据表明，MAC可能并非源自SV40。