用猿猴病毒40对猿猴病毒40转化的允许细胞进行双重感染。
Superinfection of simian virus 40-transformed permissive cells with simian virus 40.
作者信息
Barbanti-Brodano G, Swetly P, Koprowski H
出版信息
J Virol. 1970 Nov;6(5):644-51. doi: 10.1128/JVI.6.5.644-651.1970.
Abstract
Evidence that the resistance of simian virus (SV40)-transformed permissive cells to superinfection with SV40 is due to lack of virus uptake is presented. When virus uptake is enhanced, the events of infection proceed as in normal permissive cells, resulting in production of infectious virus.
摘要
有证据表明，猿猴病毒（SV40）转化的允许性细胞对SV40再次感染的抗性是由于缺乏病毒摄取。当病毒摄取增强时，感染过程与正常允许性细胞一样进行，从而产生有传染性的病毒。
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