大肠杆菌K-12核糖-5-磷酸异构酶突变体的遗传学研究。
Genetic studies on ribose 5-phosphate isomerase mutants of Escherichia coli K-12.
作者信息
Skinner A J, Cooper R A
出版信息
J Bacteriol. 1974 Jun;118(3):1183-5. doi: 10.1128/jb.118.3.1183-1185.1974.
Abstract
A gene for the constitutive ribosephosphate isomerase (rpiA) is highly cotransducible with serA at 56.2 min on the genetic linkage map of Escherichia coli K-12. Suppression of ribosephosphate isomerase A-negative mutants can occur by a regulator gene mutation permitting constitutive synthesis of the normally inducible ribosephosphate isomerase B.
摘要
在大肠杆菌K-12的遗传连锁图谱上，组成型核糖磷酸异构酶（rpiA）基因在56.2分钟处与serA基因高度共转导。核糖磷酸异构酶A阴性突变体的抑制可通过调节基因突变发生，该突变允许组成型合成通常可诱导的核糖磷酸异构酶B。
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