大肠杆菌K-12部分二倍体中紫外线抗性的显性
Dominance of ultraviolet radiation resistance in partial diploids of Escherichia coli K-12.
作者信息
Uretz R B, Markovitz A
出版信息
J Bacteriol. 1969 Nov;100(2):1118-20. doi: 10.1128/jb.100.2.1118-1120.1969.
Abstract
Although an F'13 capR(+)/capR9 strain is nonmucoid and an F'13 capR9/capR(+) strain is mucoid, both strains are ultraviolet (UV)-resistant. In contrast, haploid capR9 strains are UV-sensitive. Therefore, UV resistance is dominant to UV sensitivity, regardless of whether the capR(+) allele is on the chromosome or on the F'13 episome.
摘要
虽然F'13 capR(+)/capR9菌株是非黏液型的，而F'13 capR9/capR(+)菌株是黏液型的，但这两种菌株都对紫外线（UV）具有抗性。相比之下，单倍体capR9菌株对紫外线敏感。因此，无论capR(+)等位基因是在染色体上还是在F'13附加体上，紫外线抗性相对于紫外线敏感性都是显性的。
相似文献
1
Dominance of ultraviolet radiation resistance in partial diploids of Escherichia coli K-12.大肠杆菌K-12部分二倍体中紫外线抗性的显性
J Bacteriol. 1969 Nov;100(2):1118-20. doi: 10.1128/jb.100.2.1118-1120.1969.
2
Derepression of GDP-alpha-mannose and UDP-glucose pyrophosphorylases by a regulator gene mutation; episomal dominance in partial diploids.调节基因突变导致GDP-α-甘露糖和UDP-葡萄糖焦磷酸化酶的去阻遏；部分二倍体中的附加体显性。
Proc Natl Acad Sci U S A. 1970 Mar;65(3):625-32. doi: 10.1073/pnas.65.3.625.
3
P1-mediated transduction of a gene that controls radiation sensitivity and capsular polysaccharide synthesis from Shigella dysenteriae to Escherichia coli.P1介导的从痢疾志贺氏菌到大肠杆菌的一个控制辐射敏感性和荚膜多糖合成的基因的转导。
J Bacteriol. 1968 Jul;96(1):221-6. doi: 10.1128/jb.96.1.221-226.1968.
4
Second-site mutations in capR (lon) strains of Escherichia coli K-12 that prevent radiation sensitivity and allow bacteriophage lambda to lysogenize.大肠杆菌K-12的capR（lon）菌株中的二次位点突变可防止辐射敏感性并使噬菌体λ溶源化。
J Bacteriol. 1976 Sep;127(3):1208-16. doi: 10.1128/jb.127.3.1208-1216.1976.
5
Genetic determinant in Escherichia coli affecting thymineless death and ultraviolet sensitivity.大肠杆菌中影响无胸腺嘧啶死亡和紫外线敏感性的遗传决定因素。
J Bacteriol. 1971 Dec;108(3):1424-6. doi: 10.1128/jb.108.3.1424-1426.1971.
6
Properties of mitomycin C-sensitive mutants of Escherichia coli K-12.大肠杆菌K-12丝裂霉素C敏感突变体的特性
J Bacteriol. 1968 Feb;95(2):540-5. doi: 10.1128/jb.95.2.540-545.1968.
7
The dominance of exrB over exrB+ in heterodiploids of Escherichia coli.在大肠杆菌异源二倍体中exrB对exrB⁺的显性作用。
Genet Res. 1975 Feb;25(1):39-44. doi: 10.1017/s001667230001541x.
8
[Analysis of merodiploid strains of Sh. flexneri that have lost virulence].[对已丧失毒力的弗氏志贺菌部分二倍体菌株的分析]
Zh Mikrobiol Epidemiol Immunobiol. 1979 May(5):72-8.
9
Cloning of gene lon (capR) of Escherichia coli K-12 and identification of polypeptides specified by the cloned deoxyribonucleic acid fragment.大肠杆菌K-12基因lon（capR）的克隆及克隆的脱氧核糖核酸片段所指定的多肽的鉴定。
J Bacteriol. 1980 Aug;143(2):852-63. doi: 10.1128/jb.143.2.852-863.1980.
10
Ultraviolet-sensitive mutator strain of Escherichia coli K-12.大肠杆菌K-12的紫外线敏感突变株。
J Bacteriol. 1973 Jan;113(1):145-60. doi: 10.1128/jb.113.1.145-160.1973.
引用本文的文献
1
DNA-stimulated ATPase activity on the lon (CapR) protein.lon（CapR）蛋白上的DNA刺激的ATP酶活性。
J Bacteriol. 1984 Apr;158(1):195-201. doi: 10.1128/jb.158.1.195-201.1984.
2
Bacterial mutants defective in plasmid formation: requirement for the lon + allele.质粒形成缺陷的细菌突变体：lon + 等位基因的需求
Proc Natl Acad Sci U S A. 1971 Jul;68(7):1469-73. doi: 10.1073/pnas.68.7.1469.
3
Derepression of GDP-alpha-mannose and UDP-glucose pyrophosphorylases by a regulator gene mutation; episomal dominance in partial diploids.调节基因突变导致GDP-α-甘露糖和UDP-葡萄糖焦磷酸化酶的去阻遏；部分二倍体中的附加体显性。
Proc Natl Acad Sci U S A. 1970 Mar;65(3):625-32. doi: 10.1073/pnas.65.3.625.
4
Multiple regulator gene control of the galactose operon in Escherichia coli K-12.大肠杆菌K-12中半乳糖操纵子的多调节基因控制
J Bacteriol. 1972 Jun;110(3):1089-99. doi: 10.1128/jb.110.3.1089-1099.1972.
5
Cloning, characterization, and inactivation of the Bacillus brevis lon gene.短短芽孢杆菌lon基因的克隆、特性分析及失活
J Bacteriol. 1992 Apr;174(7):2281-7. doi: 10.1128/jb.174.7.2281-2287.1992.
6
Cloning of Escherichia coli DNA that controls cell division and capsular polysaccharide synthesis.控制大肠杆菌细胞分裂和荚膜多糖合成的DNA克隆。
Proc Natl Acad Sci U S A. 1976 Mar;73(3):697-701. doi: 10.1073/pnas.73.3.697.
7
Regulation of galactose operon at the gal operator-promoter region in Escherichia coli K-12.大肠杆菌K-12中半乳糖操纵子在半乳糖操纵基因-启动子区域的调控
J Bacteriol. 1975 May;122(2):510-7. doi: 10.1128/jb.122.2.510-517.1975.
8
Deg phenotype of Escherichia coli lon mutants.大肠杆菌lon突变体的Deg表型
J Bacteriol. 1978 Feb;133(2):844-51. doi: 10.1128/jb.133.2.844-851.1978.
本文引用的文献
1
THE EFFECT OF ACRIDINE DYES ON MATING TYPE FACTORS IN ESCHERICHIA COLI.吖啶染料对大肠杆菌交配型因子的影响
Proc Natl Acad Sci U S A. 1960 Jan;46(1):57-64. doi: 10.1073/pnas.46.1.57.
2
ANALYSIS OF A GENE CONTROLLING CELL DIVISION AND SENSITIVITY TO RADIATION IN ESCHERICHIA COLI.大肠杆菌中一个控制细胞分裂及辐射敏感性的基因分析
J Bacteriol. 1964 Mar;87(3):720-6. doi: 10.1128/jb.87.3.720-726.1964.
3
A LOCUS THAT CONTROLS FILAMENT FORMATION AND SENSITIVITY TO RADIATION IN ESCHERICHIA COLI K-12.一个控制大肠杆菌K-12中丝状形成和对辐射敏感性的基因座。
Genetics. 1964 Feb;49(2):237-46. doi: 10.1093/genetics/49.2.237.
4
REGULATORY MECHANISMS FOR SYNTHESIS OF CAPSULAR POLYSACCHARIDE IN MUCOID MUTANTS OF ESCHERICHIA COLI K12.大肠杆菌K12黏液突变体中荚膜多糖合成的调控机制
Proc Natl Acad Sci U S A. 1964 Feb;51(2):239-46. doi: 10.1073/pnas.51.2.239.
5
RADIOSENSITIVITY IN ESCHERICHIA COLI.大肠杆菌的放射敏感性
J Gen Microbiol. 1963 Nov;33:283-92. doi: 10.1099/00221287-33-2-283.
6
Suppression of radiation sensitivity and capsular polysaccharide synthesis in Escherichia coli K-12 by ochre suppressors.赭石抑制基因对大肠杆菌K-12辐射敏感性和荚膜多糖合成的抑制作用
J Bacteriol. 1967 Aug;94(2):388-95. doi: 10.1128/jb.94.2.388-395.1967.
7
Induction of capsular polysaccharide synthesis by rho-fluorophenylalanine in Escherichia coli wild type and strains with altered phenylalanyl soluble ribonucleic acid synthetase.在大肠杆菌野生型及苯丙氨酰可溶性核糖核酸合成酶发生改变的菌株中，ρ-氟苯丙氨酸对荚膜多糖合成的诱导作用。
J Bacteriol. 1967 Feb;93(2):584-91. doi: 10.1128/jb.93.2.584-591.1967.
8
Conditional mutations involving septum formation in Escherichia coli.涉及大肠杆菌中隔膜形成的条件突变。
J Bacteriol. 1967 Jan;93(1):107-14. doi: 10.1128/jb.93.1.107-114.1967.
9
Repair of radiation-induced damage to the cell division mechanism of Escherichia coli.大肠杆菌细胞分裂机制辐射损伤的修复
J Bacteriol. 1966 Feb;91(2):737-42. doi: 10.1128/jb.91.2.737-742.1966.
10
A regulator gene that is dominant on an episome and recessive on a chromosome.一种调节基因，它在附加体上是显性的，而在染色体上是隐性的。
Proc Natl Acad Sci U S A. 1965 Oct;54(4):1084-91. doi: 10.1073/pnas.54.4.1084.
Zotero 插件