Tessman I
Science. 1968 Aug 2;161(3840):481-2. doi: 10.1126/science.161.3840.481.
In certain recombination-deficient (Rec(-)) bacterial strains genetic recombination of bacteriophage S13 is reduced, but the existence of some residual recombination has suggested that there is a secondary mechanism of phage recombination that is still functioning. In these Rec(-)strains it is found that there is no stimulation of recombination by irradiation of the parental phage with ultraviolet light, in contrast to the large increase found when irradiated phage particles infect a Rec(+) host. This selective stimulation of phage recombination in the Rec(+) but not in the Rec(-) strains supports the view that the phage uses at least two mechanisms of genetic recombination.
在某些重组缺陷（Rec(-)）细菌菌株中，噬菌体S13的基因重组减少，但一些残余重组的存在表明存在一种仍在起作用的噬菌体重组二级机制。在这些Rec(-)菌株中发现，用紫外线照射亲本噬菌体不会刺激重组，这与照射后的噬菌体颗粒感染Rec(+)宿主时重组大幅增加形成对比。在Rec(+)菌株而非Rec(-)菌株中对噬菌体重组的这种选择性刺激支持了噬菌体至少使用两种基因重组机制的观点。
