Hausman S Z, Thompson J, London J
J Bacteriol. 1984 Oct;160(1):211-5. doi: 10.1128/jb.160.1.211-215.1984.
A futile xylitol cycle appears to be responsible for xylitol-mediated inhibition of growth of Lactobacillus casei Cl-16 at the expense of ribitol. The gratuitously induced xylitol-specific phosphoenolpyruvate-dependent phosphotransferase accumulates the pentitol as xylitol-5-phosphate, a phosphatase cleaves the latter, and an export system expels the xylitol. Operation of the cycle rapidly dissipates the ribitol-5-phosphate pool (and ultimately the energy supply of the cell), thereby producing bacteriostasis.
一个无效的木糖醇循环似乎是木糖醇介导的干酪乳杆菌Cl-16生长抑制的原因，这种抑制是以核糖醇为代价的。无端诱导的木糖醇特异性磷酸烯醇丙酮酸依赖性磷酸转移酶将戊糖醇积累为木糖醇-5-磷酸，一种磷酸酶切割后者，并且一个输出系统排出木糖醇。该循环的运行迅速消耗核糖醇-5-磷酸池（并最终消耗细胞的能量供应），从而产生抑菌作用。
