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2
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Studies on the pathogenesis of influenza virus pneumonia in mice.小鼠流感病毒肺炎发病机制的研究。
J Pathol Bacteriol. 1962 Jan;83:207-17.
2
Concurrent respiratory syncytial virus and influenza A infections in the institutionalized elderly and chronically ill.机构养老老人及慢性病患者中呼吸道合胞病毒与甲型流感病毒的合并感染
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Variability of results of lymphocyte transformation assays in normal human volunteers. Responses of mononuclear leukocytes to mitogen stimulation.正常人类志愿者淋巴细胞转化试验结果的变异性。单核白细胞对有丝分裂原刺激的反应。
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Pathogenicity of influenza virus.流感病毒的致病性。
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Interaction of influenza virus with mouse macrophages.流感病毒与小鼠巨噬细胞的相互作用。
Infect Immun. 1981 Feb;31(2):751-7. doi: 10.1128/iai.31.2.751-757.1981.
6
Interferon production by human mononuclear leukocytes: differences between respiratory syncytial virus and influenza viruses.人单核白细胞产生干扰素：呼吸道合胞病毒与流感病毒之间的差异。
Infect Immun. 1981 Apr;32(1):300-3. doi: 10.1128/iai.32.1.300-303.1981.
7
Mitogen responses and interferon production after exposure of human macrophages to infectious and inactivated influenza viruses.人类巨噬细胞暴露于感染性和灭活流感病毒后有丝分裂原反应和干扰素产生
J Med Virol. 1980;5(1):17-23. doi: 10.1002/jmv.1890050103.
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Effect of virus infections on the function of the immune system.病毒感染对免疫系统功能的影响。
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Inactivation of influenza virus by human lymphocytes.人淋巴细胞对流感病毒的灭活作用。
Clin Exp Immunol. 1974 Apr;16(4):583-91.
10
Replication or inactivation of different viruses by human lymphocyte preparations.人淋巴细胞制剂对不同病毒的复制或灭活作用。
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流感病毒、呼吸道合胞病毒和仙台病毒对人淋巴细胞和巨噬细胞的不同影响。

Different effects of influenza virus, respiratory syncytial virus, and Sendai virus on human lymphocytes and macrophages.

作者信息

Roberts N J

出版信息

Infect Immun. 1982 Mar;35(3):1142-6. doi: 10.1128/iai.35.3.1142-1146.1982.

DOI:10.1128/iai.35.3.1142-1146.1982
PMID:6175576
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC351166/
Abstract

Influenza virus, respiratory syncytial virus, and Sendai virus depress human cell-mediated immune responses, such as mitogen-induced lymphocyte transformation, but differ in their ability to induce other immune defense mechanisms, such as interferon production. Exposure to the different viruses resulted in depressed transformation responses to the mitogen phytohemagglutinin by affecting the function of lymphocytes, or macrophages, or both cell types.

摘要

流感病毒、呼吸道合胞病毒和仙台病毒会抑制人类细胞介导的免疫反应，如丝裂原诱导的淋巴细胞转化，但它们在诱导其他免疫防御机制（如干扰素产生）的能力上有所不同。接触不同病毒会通过影响淋巴细胞或巨噬细胞或这两种细胞类型的功能，导致对丝裂原植物血凝素的转化反应受到抑制。